AI Summary & Key Takeaways
AI InsightIn this article a case with hyperhomocysteinemia formated recurrent thrombosis, who has presented to hospital with acute myocardial infarction and ischemic stroke is reported.
📌 3 Key Takeaways
Abstract
Hyperhomocysteinemia is an independent risk factor for atherosclerosis and increases the risk of coronary arterial heart disease and ischemic cerebrovascular event. Gene defects of enzyms or nutritional insufficiency of cofactor vitamins in homocystein metabolism are the main etiology of hyperhomocysteinemia. Hyperhomocysteinemia is associated with the increase in development of atherosc- lerosis and also thrombosis. In this article a case with hyperhomocysteinemia formated recurrent thrombosis, who has presented to hospital with acute myocardial infarction and ischemic stroke is reported.
Özet
Hiperhomosisteinemi ateroskleroz için bağımsız bir risk faktörü olup, hem kardiyovasküler hem de serebrovasküler olay riskini arttırır. Etyolojisinde ya homosistein metabolizmasında yer alan enzimlerin genetik defektleri ya da kofaktör olarak kullandıkları vitaminlerin nütrisyonel eksiklikleri yatmaktadır. Hiperhomosisteinemi hem ateroskleroz hem de tromboz gelişiminde artışla ilişkilidir. Bu yazıda akut miyokard enfarktüsü ve akut iskemik inme tablosunda başvurup, tekrarlayan tromboz ile seyreden hiperhomosisteinemili bir olgu bildirildi.