Dergiler / Iğdır Üniversitesi Fen Bilimleri Enstitüsü Dergisi / 2026 / Cilt: 15 Sayı: 2

Nano-Silika Katkılı Kenevir/Reçine Kompozitleri Suda Yaşlandırmanın Darbe Dayanımına Etkisi

The Effect of Water Aging on the Impact Strength of Nano-Silica HempThe Effect of Water Aging on the Impact Strength of Nano-Silica Hemp Fiber/Epoxy CompositesFiber/Epoxy Composites.

Sayfa
682–691
Yayın Tarihi
2025-06-01
🏛️Bu makalenin bibliyografik verileri ve tam metin bağlantısı DergiPark üzerinden sağlanmıştır.
DergiPark Kaydı ↗
✨

Yapay Zeka Hızlı Bakış & Çıkarımlar

AI Insight
⚡ TL;DR(1 Cümlede Hızlı Çıkarım)

Yaşlandırma sonrasında ise darbe enerjisinde en fazla düşüş %1.5 nano oranında %33.4 olarak ölçülürken toklukta %3 nano oranında %32 olarak ölçülmüştür.

📌 3 Maddede Bu Makale

🎯
Amaç & Hipotez:Bu çalışmada doğal kenevir elyaf kumaş ile üretilen nano silika katkılı polimer kompozitlerin su ile yaşlandırılmasının Charpy darbe davranışına etkileri araştırılmıştır.
📊
Temel Bulgular & Veri:Nano parçacık katkısının hidrofilik davranışa etkilerini gözlemlemek amacıyla nano silika parçacıkları ağırlıkça %0.5, %1.5 ve %3 oranlarında reçineye eklenerek kompozit üretimleri gerçekleştirilmiştir.
💡
Sonuç & Katkı:Yaşlandırma sonrasında ise darbe enerjisinde en fazla düşüş %1.5 nano oranında %33.4 olarak ölçülürken toklukta %3 nano oranında %32 olarak ölçülmüştür.
🔬Tür:Araştırma Makalesi
📍Kapsam:Genişletilmiş Akademik Örneklem
⚙️Değişkenler:Kenevir elyaf kompozit · Nano silika · Suda yaşlandırma

Özet

Bu çalışmada doğal kenevir elyaf kumaş ile üretilen nano silika katkılı polimer kompozitlerin su ile yaşlandırılmasının Charpy darbe davranışına etkileri araştırılmıştır. Nano parçacık katkısının hidrofilik davranışa etkilerini gözlemlemek amacıyla nano silika parçacıkları ağırlıkça %0.5, %1.5 ve %3 oranlarında reçineye eklenerek kompozit üretimleri gerçekleştirilmiştir. Üretilen kompozitlerden ISO 179/92 standardına göre 55 x 10 mm ölçüsünde numuneler kesilmiş ve 800 saat 30℃’de çeşme suyunda yaşlandırma işlemi gerçekleştirilmiştir. Yaşlandırılan numuneler 15 J sarkaç enerjili Köger 3/70 darbe test cihazı ile test edilmiştir. Ölçülen sonuçlar hem emilen enerji hem de tokluğu gösterecek şekilde tablo haline getirilmiştir. Testler sonucunda nano katkısının tokluğu ve emilen enerjiyi düşürdüğü görülmüştür. %1.5 nano silikanın tokluğu %33.73 emilen enerjiyi ise %22,54 azalttığı gözlenmiştir. Yaşlandırma sonrasında ise darbe enerjisinde en fazla düşüş %1.5 nano oranında %33.4 olarak ölçülürken toklukta %3 nano oranında %32 olarak ölçülmüştür.

Abstract

In this study, the effects of water aging on the Charpy impact behavior of polymer composites reinforced with natural hemp fiber fabric and nano-silica were investigated. To observe the influence of nano-particle addition on hydrophilic behavior, nano-silica particles were added to the resin in weight percentages of 0.5%, 1.5%, and 3% for composite production. Samples with dimensions of 55 x 10 mm were cut from the produced composites according to ISO 179/92 standard, and the aging process was carried out in tap water at 30℃ for 800 hours. The aged samples were tested using a Köger 3/70 impact test device with 15 J pendulum energy. The measured results were tabulated to show both absorbed energy and toughness. The test results revealed that the addition of nano-silica reduced both toughness and absorbed energy. It was observed that 1.5% nano-silica reduced toughness by 33.73% and absorbed energy by 22.54%. After aging, the greatest decrease in impact energy was measured as 33.4% for the 1.5% nano-silica, while the greatest reduction in toughness was 32% for the 3% nano-silica.

🔖

Bu Makaleye Atıf Yap

Erkliğ, A., Yıldırım, B. (2025). Nano-Silika Katkılı Kenevir/Reçine Kompozitleri Suda Yaşlandırmanın Darbe Dayanımına Etkisi. Iğdır Üniversitesi Fen Bilimleri Enstitüsü Dergisi, Cilt: 15 Sayı: 2, 682–691. https://doi.org/10.21597/jist.1562248

📄 Tam Metin PDF Okuyucu

📄

PDF görüntüleyici hazırlanıyor...

Kaynakça (41)

  1. Adzhubei, I., Jordan, D. M., & Sunyaev, S. R. (2013). Predicting functional effect of human missense mutations using PolyPhen‐2. Current Protocols in Human Genetics, 76(1), 7–20. https://doi.org/10.1002/0471142905.hg0720s76
  2. Avivi, A., Albrecht, U., Oster, H., Joel, A., Beiles, A., & Nevo, E. (2001). Biological clock in total darkness: The Clock/MOP3 circadian system of the blind subterranean mole rat. Proceedings of the National Academy of Sciences, 98(24), 13751-13756. https://doi.org/10.1073/pnas.181484498
  3. Civelek, İ., Kankılıç, T., & Akın, D. F. (2024). An investigation of Clock gene variations in Turkish Nannospalax species. Russian Journal of Genetics, 60(5), 626-639. https://doi.org/10.1134/S1022795424040057
  4. David-Gray, Z. K., Cooper, H. M., Janssen, J. W. H., Nevo, E., & Foster, R. G. (1999). Spectral tuning of a circadian photopigment in a subterranean ‘blind’ mammal (Spalax ehrenbergi). FEBS Letters, 461(3), 343–347. https://doi.org/10.1016/S0014-5793(99)01455-6
  5. Dehdari Ebrahimi, N., Sadeghi, A., Shojaei-Zarghani, S., Shahlaee, M. A., Taherifard, E., Rahimian, Z.,. & Safarpour, A. R. (2023). Protective effects of exogenous melatonin therapy against oxidative stress to male reproductive tissue caused by anti-cancer chemical and radiation therapy: A systematic review and meta-analysis of animal studies. Frontiers in Endocrinology, 14, 1184745. https://doi.org/10.3389/fendo.2023.1184745
  6. Doyle, J. (1991). DNA protocols for plants. In Molecular techniques in taxonomy (pp. 283-293). Berlin, Heidelberg: Springer Berlin Heidelberg.
  7. Dubocovich, M. L. (2007). Melatonin receptors: Role on sleep and circadian rhythm regulation. Sleep Medicine, 8, 34-42. https://doi.org/10.1016/j.sleep.2007.10.007
  8. European Bioinformatics Institute (EMBL-EBI). (n.d.). Ensembl Genome Browser [Veritabanı]. https://www.ensembl.org (Erişim: Haziran, 2024).
  9. Flesher, J. W., Horn, J., & Lehner, A. F. (1998). Carcinogenicity of 1-hydroxy-3-methylcholanthrene and its electrophilic sulfate ester 1-sulfooxy-3-methylcholanthrene in Sprague-Dawley rats. Biochemical and Biophysical Research Communications, 243(1), 30–35. https://doi.org/10.1006/bbrc.1997.8048
  10. Foulkes, N. S., Borjigin, J., & Snyder, S. H. (1997). Rhythmic transcription: The molecular basis of circadian melatonin synthesis. Trends in Neurosciences, 20(10), 487-492. https://doi.org/10.1016/S0166-2236(97)01109-0
  11. Gao, Y., Wu, X., Zhao, S., Zhang, Y., Ma, H., Yang, Z.,. & Zhang, Q. (2019). Melatonin receptor depletion suppressed hCG-induced testosterone expression in mouse Leydig cells. Cellular & Molecular Biology Letters, 24(1), 1-14.
  12. Gorbunova, V., Hine, C., Tian, X., Ablaeva, J., Gudkov, A. V., Nevo, E., & Seluanov, A. (2012). Cancer resistance in the blind mole rat is mediated by concerted necrotic cell death mechanism. Proceedings of the National Academy of Sciences, 109(47), 19392–19396. https://doi.org/10.1073/pnas.1217211109
  13. Hall, T. A. (1999). BioEdit: A user-friendly biological sequence alignment editor and analysis program for Windows 95/98/N. Nucleic Acids Symposium Series, Oxford, 95-98.
  14. Hannibal, J., Hindersson, P., Nevo, E., & Fahrenkrug, J. (2002). The circadian photopigment melanopsin is expressed in the blind subterranean mole rat, Spalax. Neuroreport, 13(11), 1411-1414.
  15. Higgins, E. M., Bos, J. M., Mason-Suares, H., Tester, D. J., Ackerman, J. P., MacRae, C. A.,. & Ackerman, M. J. (2017). Elucidation of MRAS-mediated Noonan syndrome with cardiac hypertrophy. JCI Insight, 2(5).
  16. Human Genome Mutation Database. (n.d.). PolyPhen-2: Prediction of functional effects of human nsSNPs. http://genetics.bwh.harvard.edu/pph2/ (Erişim: Haziran, 2024).
  17. Jimenez‐Jorge, S., Guerrero, J. M., Jimenez‐Caliani, A. J., Naranjo, M. C., Lardone, P. J., Carrillo‐Vico, A.,. & Molinero, P. (2007). Evidence for melatonin synthesis in the rat brain during development. Journal of Pineal Research, 42(3), 240-246. https://doi.org/10.1111/j.1600-079X.2006.00411.x
  18. Jung‐Hynes, B., Reiter, R. J., & Ahmad, N. (2010). Sirtuins, melatonin and circadian rhythms: Building a bridge between aging and cancer. Journal of Pineal Research, 48(1), 9-19. https://doi.org/10.1111/j.1600-079X.2009.00729.x
  19. Liu, J., Clough, S. J., Hutchinson, A. J., Adamah-Biassi, E. B., Popovska-Gorevski, M., & Dubocovich, M. L. (2016). MT1 and MT2 melatonin receptors: A therapeutic perspective. Annual Review of Pharmacology and Toxicology, 56(1), 361-383. https://doi.org/10.1146/annurev-pharmtox-010814-124742
  20. Ma, Z., Xu, L., Liu, D., Zhang, X., Di, S., Li, W.,. & Yan, X. (2020). Utilizing melatonin to alleviate side effects of chemotherapy: A potentially good partner for treating cancer with aging. Oxidative Medicine and Cellular Longevity, 2020(1), 6841581. https://doi.org/10.1155/2020/6841581
  21. Manov, I., Hirsh, M., Iancu, T. C., Malik, A., Sotnichenko, N., Band, M., Avivi, A., & Shams, I. (2013). Pronounced cancer resistance in a subterranean rodent, the blind mole-rat, Spalax: In vivo and in vitro evidence. BMC Biology, 11(1), 1–18. https://doi.org/10.1186/1741-7007-11-91
  22. Najafi, M., Shirazi, A., Motevaseli, E., Geraily, G., Norouzi, F., Heidari, M., & Rezapoor, S. (2017). The melatonin immunomodulatory actions in radiotherapy. Biophysical Reviews, 9, 139-148. https://doi.org/10.1007/s12551-017-0256-8
  23. National Center for Biotechnology Information. (n.d.). Basic Local Alignment Search Tool (BLAST) [Veritabanı]. National Institutes of Health. https://blast.ncbi.nlm.nih.gov/Blast.cgi (Erişim: Haziran, 2024).
  24. National Center for Biotechnology Information. (2024). NCBI Genome Database. https://www.ncbi.nlm.nih.gov/genome/ (Erişim: Haziran, 2024).
  25. Németh, A., Hegyeli, Z., Sendula, T., Horváth, M., Czabán, D., & Csorba, G. (2016). Danger underground and in the open–predation on blind mole rats (Rodentia: Spalacinae) revisited. Mammal Review, 46(3), 204-214.
  26. Nevo, E. (2022). Evolution of communication systems underground in a blind mammal, Spalax. In P. S. M. Hill, V. Mazzoni, N. Stritih-Peljhan, M. Virant-Doberlet, & A. Wessel (Eds.), Biotremology: Physiology, Ecology, and Evolution (Vol. 8). Springer, Cham. https://doi.org/10.1007/978-3-030-97419-0_15
  27. Pham, T. T., Lee, E. S., Kong, S. Y., Kim, J., Kim, S. Y., Joo, J.,. & Park, B. (2019). Night-shift work, circadian and melatonin pathway-related genes and their interaction on breast cancer risk: Evidence from a case-control study in Korean women. Scientific Reports, 9(1), 10982.
  28. Steele, T. A., St Louis, E. K., Videnovic, A., & Auger, R. R. (2021). Circadian rhythm sleep–wake disorders: A contemporary review of neurobiology, treatment, and dysregulation in neurodegenerative disease. Neurotherapeutics, 18(1), 53–74. https://doi.org/10.1007/s13311-021-01031-8
  29. Sun, H., Pan, D., Liu, D., Cheng, Y., Zhang, Y., & Wang, Z. (2022). Melatonin secretion, molecular expression and evolution of MT1/2 in two Lasiopodomys species. Mammalian Biology, 102(1), 99-107.
  30. The UniProt Consortium. (n.d.). UniProt Knowledgebase [Veritabanı]. European Bioinformatics Institute. https://www.uniprot.org (Erişim: Haziran, 2024).
  31. Topachevskii, W. A. (1969). Fauna USSR Spalacidae. Leningrad, USSR: Nauka (English translation: Springfield, VA, USA: US Department of Commerce National Technical Information Service).
  32. University of Manchester. (n.d.). PRINTS Database [Veritabanı]. https://www.bioinf.manchester.ac.uk/dbbrowser/PRINTS/ (Erişim: Haziran, 2024).
  33. Wang, X. (2009). The antiapoptotic activity of melatonin in neurodegenerative diseases. CNS Neuroscience & Therapeutics, 15(4), 345-357. https://doi.org/10.1111/j.1755-5949.2009.00105.x
  34. Zhang, J., Jiang, H., Du, K., Xie, T., Wang, B., Chen, C.,. & Yuan, Y. (2021). Pan‐cancer analyses reveal genomics and clinical characteristics of the melatonergic regulators in cancer. Journal of Pineal Research, 71(3), e12758. https://doi.org/10.1111/jpi.12758
  35. Dehdari Ebrahimi, N., Sadeghi, A., Shojaei-Zarghani, S., Shahlaee, M. A., Taherifard, E., Rahimian, Z.,. & Safarpour, A. R. (2023). Protective effects of exogenous melatonin therapy against oxidative stress to male reproductive tissue caused by anti-cancer chemical and radiation therapy: A systematic review and meta-analysis of animal studies. Frontiers in Endocrinology, 14, 1184745. https://doi.org/10.3389/fendo.2023.1184745
  36. Gao, Y., Wu, X., Zhao, S., Zhang, Y., Ma, H., Yang, Z.,. & Zhang, Q. (2019). Melatonin receptor depletion suppressed hCG-induced testosterone expression in mouse Leydig cells. Cellular & Molecular Biology Letters, 24(1), 1-14.
  37. Higgins, E. M., Bos, J. M., Mason-Suares, H., Tester, D. J., Ackerman, J. P., MacRae, C. A.,. & Ackerman, M. J. (2017). Elucidation of MRAS-mediated Noonan syndrome with cardiac hypertrophy. JCI Insight, 2(5).
  38. Jimenez‐Jorge, S., Guerrero, J. M., Jimenez‐Caliani, A. J., Naranjo, M. C., Lardone, P. J., Carrillo‐Vico, A.,. & Molinero, P. (2007). Evidence for melatonin synthesis in the rat brain during development. Journal of Pineal Research, 42(3), 240-246. https://doi.org/10.1111/j.1600-079X.2006.00411.x
  39. Ma, Z., Xu, L., Liu, D., Zhang, X., Di, S., Li, W.,. & Yan, X. (2020). Utilizing melatonin to alleviate side effects of chemotherapy: A potentially good partner for treating cancer with aging. Oxidative Medicine and Cellular Longevity, 2020(1), 6841581. https://doi.org/10.1155/2020/6841581
  40. Pham, T. T., Lee, E. S., Kong, S. Y., Kim, J., Kim, S. Y., Joo, J.,. & Park, B. (2019). Night-shift work, circadian and melatonin pathway-related genes and their interaction on breast cancer risk: Evidence from a case-control study in Korean women. Scientific Reports, 9(1), 10982.
  41. Zhang, J., Jiang, H., Du, K., Xie, T., Wang, B., Chen, C.,. & Yuan, Y. (2021). Pan‐cancer analyses reveal genomics and clinical characteristics of the melatonergic regulators in cancer. Journal of Pineal Research, 71(3), e12758. https://doi.org/10.1111/jpi.12758