Journals / ADO Klinik Bilimler Dergisi / 2009 / Cilt: 3 - Sayı: 2

The role of bacteria in pulpal diseases

Pulpa Hastalıklarında Bakterilerin Rolü

Pages
374–379
Publication Date
2009-07-01
DOI
—
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AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

Discovery of the microorganisms that play roles in endodontic enfections and their relationship with each other in mixed infections point to the close relationship between basic science and clinical practise in the special field of endodontics.Discovering of these microorganisms, including their destructive potential, their requirements for growth, their routes of entry, their sensitivity to pharmacotherapeutic agents, their synergistic dependence on each other, and their relationship to clinical symptoms is necessary to formulate a new approach to root canal therapy

📌 3 Key Takeaways

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Objective & Hypothesis:It was accepted that without bacterial involvement, only minor inflammation occured in exposed pulps.
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Key Findings & Data:Pulpal abcesses, periapical periontitis, swelling, and pain are the result of mixed microbial infections.
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Conclusion & Impact:Discovery of the microorganisms that play roles in endodontic enfections and their relationship with each other in mixed infections point to the close relationship between basic science and clinical practise in the special field of endodontics.Discovering of these microorganisms, including their destructive potential, their requirements for growth, their routes of entry, their sensitivity to pharmacotherapeutic agents, their synergistic dependence on each other, and their relationship to clinical symptoms is necessary to formulate a new approach to root canal therapy
🔬Design:Research Article
📍Scope:Comprehensive Sample
⚙️Variables:Diş aşınması · Restorasyon · Estetik

Abstract

It was accepted that without bacterial involvement, only minor inflammation occured in exposed pulps. Pulpal abcesses, periapical periontitis, swelling, and pain are the result of mixed microbial infections. Discovery of the microorganisms that play roles in endodontic enfections and their relationship with each other in mixed infections point to the close relationship between basic science and clinical practise in the special field of endodontics.Discovering of these microorganisms, including their destructive potential, their requirements for growth, their routes of entry, their sensitivity to pharmacotherapeutic agents, their synergistic dependence on each other, and their relationship to clinical symptoms is necessary to formulate a new approach to root canal therapy

Özet

Bakteriler olmadan, ekspoze pulpada sadece hafif düzeyde enflamasyonların oluştuğu kabul edilmiş bir gerçektir. Pulpal apseler, periapikal periodontitisler, periapikal şişlik ve ağrı; karma mikrobiyal enfeksiyonlar sonucu oluşmaktadır. Endodontik enfeksiyonlarda rol oynayan mikroorganizmalar ve karma enfeksiyonlarda bunların birbiriyle olan ilişkilerinin keşfedilmesi bir çok araştırmanın konusu olmuştur.Bu mikroorganizmaların yıkım potansiyellerinin, üremeleri için gereksinimlerinin, giriş yollarının, ilaçlara karşı hassasiyetlerinin, birbirleriyle olan sinerjistik etkileşimlerinin ve klinik semptomlarla olan ilişkilerinin anlaşılması; kök kanal tedavisine yeni yaklaşımların getirilmesini gerektirmektedir

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Cite This Article

TİRALİ, R. E., BODUR, H. (2009). The role of bacteria in pulpal diseases. ADO Klinik Bilimler Dergisi, Cilt: 3 - Sayı: 2, 374–379. https://ajindex.com/articles/728936

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References (10)

  1. Seltzer S. Endodontology: Biologic Considerations in Endodontic Procedures. 2nd ed. Philadelphia, Lea and Febiger, 1988, 326-344. Aydın M. Endodontik Mikrobiyoloji: Alaçam T. ed. Endodonti 2. baskı. Ankara: Fakülteler Kitapevi Barıs Yayınları, 2000, 313–384. Baumgartner JC. Endodontic Microbiology: Wal- ton RE, Torabinejad M. editors. Principles and practise of Endodontics. 3rd ed. Philadelphia: W.B. Saunders Company, 2002, 282-294. Morse DR. Microbiology and pharmacology: Co- hen S, Burns RC,editors. Pathways of the pulp. 2nd ed. St.Louis: The C.V. Mosby Company, 1980, 321-339.
  2. Pisano JV, Weine FS. Microbiology of endodontics:Weine FS, editor. Endodontic The- rapy. 5th. ed. St. Louis:Mosby Year Book Inc., 1996, 693-712.
  3. Goodman AD, Nisengard RJ. Periapical Infecti- ons: Newman MG, Nisengard RJ, editors. Oral Microbiology and Immunology. 1st ed. Philadelp- hia: W.B. Saunders Company, 1988, 447-454. Bergenholtz G.Inflammatory response of the dental pulp to bacterial irritation. J Endod.7:100,1981. Cvek M. A clinical report on partial pulpotomy and capping with calcium hydroxide in perma- nent incisors with complicated crown fractures. J Endod.4: 232-7,1978.
  4. Delivanis PD, Fan VS. The localization of blood- borne bacteria in instrumented unfilled and over instrumented canals. J Endod. 10: 521-524, 1984. Sundqvist G. Associations between microbial spe- cies in dental root canal infections. Oral Microbiol Immunol.7:257,1992.
  5. Dahlen G, Haapasalo M. Microbiology of apical periodontitis. In:Ørstavik D, Ford TRP, editors. Es- sential Endodontology Prevention and Treatment of Apical Periodontitis. 1st ed. Oxford: Blackwell Publihing Company, 1998, 106-130.
  6. Seltzer S, Farber PA. Microbiologic factors in en- dodontology. Oral Surg Oral Med Oral Pathol. 78: 634-645, 1994.
  7. Tomic-Karovic K, Jelinek E.Comparative study of the bacterial flora in the surrounding, the root ca- nals and sockets of deciduous molars. Int Dent J. 21:375-88, 1971. Schuster GS. Oral Microbiology and Infectious Di- sease. 3rd ed. Philadelphia: B.C.Decker Inc.,1990, 562-548. Ang Ö. Agız Mikrobiyolojisi. İstanbul: Nobel Tıp Kitapevi, 1990, 359-379.
  8. Ryan KJ, Ray CG. Ed. Sherris Medical Microbio- logy. 4th ed. Mc Graw Hill, 2004,294-5.
  9. Borssen E, Sundqvist G. Actinomyces of infected dental root canals. Oral Surg Oral Med Oral Pat- hol. 51: 643-648, 1981.
  10. Hawser SP, Douglas LJ. Biofilm formation by Can- dida species on the surface of catheter materials in vitro. Infect Immun. 62: 915-921, 1994. Van Win