Journals / Retina-Vitreus / 2004 / Cilt: 12 - Sayı: 1

Clinical and fluorescein angiographic features in cases with ocular ischemic syndrome

Oküler iskemik sendromlu olgularda klinik ve flöresein anjiografik bulgular

Pages
37–41
DOI
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AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

Conclusion: If is very important fo diagnose fhe OİS and fo recognise significantly compromised carofid circulation in most cases of OİS, so life threatening neurological or cardiovascular complications can be prevenfed in this sfudy, we stressed the variefy of clinical and angiographic findings in cases wifh OİS.

📌 3 Key Takeaways

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Objective & Hypothesis:Purpose: To evaluate the clinical and fluorescein angiographic (FA) findings in the cases with 'Ocular ischemic syndrome'(QIS) Material and Methods: Seventeen eyes of 14 cases with OİS were included fhis study.
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Key Findings & Data:Results: Rubeosis iridis was determined in 8 eyes with OİS (47.1%) and 2 eyes had neovascular glaucoma (71.8%).
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Conclusion & Impact:Conclusion: If is very important fo diagnose fhe OİS and fo recognise significantly compromised carofid circulation in most cases of OİS, so life threatening neurological or cardiovascular complications can be prevenfed in this sfudy, we stressed the variefy of clinical and angiographic findings in cases wifh OİS.
🔬Design:Research Article
📍Scope:Comprehensive Sample
⚙️Variables:Göz hastalıkları · Floresan anjiyografi · Görme keskinliği

Abstract

Purpose: To evaluate the clinical and fluorescein angiographic (FA) findings in the cases with 'Ocular ischemic syndrome'(QIS) Material and Methods: Seventeen eyes of 14 cases with OİS were included fhis study. The measurements of best corrected visual acuity, anterior and posterior segment examinations of cases with OİS were performed and FA was recorded. Results: Rubeosis iridis was determined in 8 eyes with OİS (47.1%) and 2 eyes had neovascular glaucoma (71.8%). Fundus examinafion was revealed midperipheral retinal haemorrhages in all eyes with OİS. Retinal venous dilatation without tortuosity was found in 9 eyes, cherry red spot in the macular area was seen in 4 eyes. There was retinal neovascularization in one eye, cotton wool spot in one eye and microaneurysms in 7 eyes. Anterior ischemic opfic neuropafhy, optic disk pale, optic disk neovascularizafion, retinal arterial sclerosis and narrovving, fhe findings due to sequel of retinal artery occlusion, intraarterial embolic plaque were the another findings of posterior segment. The fluorescein angiographic signs of OİS included patchy choroidal filling in 5 eyes, increased retinal arteriovenous circulation time in 8 eyes, delayed arm-retina time in 9 eyes, areas of retinal capillary non-perfusion in 5 eyes. Conclusion: If is very important fo diagnose fhe OİS and fo recognise significantly compromised carofid circulation in most cases of OİS, so life threatening neurological or cardiovascular complications can be prevenfed in this sfudy, we stressed the variefy of clinical and angiographic findings in cases wifh OİS.

Özet

Amaç: Oküler iskemik sendromlu (OİS) olgularda klinik ye flöresein anjiografik (FA) bulguların değerlendirilmesi Gereç ve Yöntem: OİS tanısı konulan, 14 olgunun 17 gözü çalışmaya alındı. Olguların tümünün eri iyi düzeltilmiş görme keskinlikleri ölçüldü, detaylı ön ve arka segmenf muayeneleri yapıldı ve FA çekildi. Bulgular: OİS'lu 8 gözde (%47.1) rubeozis iridis saptandı, 2'sinde (%11.8) neovasküler glokom olduğu belirlendi. Göz dibi muayenesinde tüm gözlerde midperiferde retinal hemoraji saptandı. Dokuz gözde retinal venlerde dilafasyon ve düzleşme, 4 gözde- makulada 'cherry red spot' saptandı. Bir gözde retinal neovaskülarizasyon, bir gözde yumuşak elcsuda, 7 gözde mikroanevrizma bulunduğu gözlendi. Anterior iskemik optik nöropati, optik disk solukluğu, optik disk neovaskülarizasyonu, retinal arterlerde incelme, skleroz, geçirilmiş santral retinal arter tıkanıklığına ait değişiklikler, embolik plak varlığı diğer arka segmenf bulgularıydı. FA bulguları, 5 gözde lekeli koroid dolumu, 8 gözde uzamış arteriovenöz geçiş zamanı, 9 gözde kol-retina zamanında uzama, 5 gözde kopiller nonperfüzyon idi. Sonuç: OİS tanısı ve çoğu olguda önemli ölçüde etkilenmiş olan karotis dolaşımının belirlenmesi çok önemlidir. Böylelikle, - OIS'lu hastalarda yaşamı tehdit eden nörolojik ve kardiovasküler komplikasyonlar önlenebilir. Bu çalışmada, OİS tanısını koyduracak klinik ve anjiografik bulguların çeşitliliği vurgulanmıştır.

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Cite This Article

MENTEŞ, J., AFRASHI, F., AZARSIZ, S., ERAKGÜN, T., AKKIN, C. (2004). Clinical and fluorescein angiographic features in cases with ocular ischemic syndrome. Retina-Vitreus, Cilt: 12 - Sayı: 1, 37–41. https://ajindex.com/articles/307979

References (19)

  1. Knox DL.: Ischémie ocular inflammation. Am J Ophthalmol 1965;60:995-1002
  2. Malhotra R, Evans KG.: Management of ocular ischémie syndrome. Br J Ophthalmol 2000;84: 1428-1431
  3. Gönder JR. The ocular ischémie syndrome. Can J Ophthalmol T986;21:1-2
  4. Brown GC: Ocular ischémie syndrome. In: Ryan SJ (ed) Retina, Medical Retina. The CV Mosby Co, St Louis, 1995; 1515-1527
  5. Carter JE. Chronic ocular ischemia and carotid vascular disease. Stroke 1985;16:721-728
  6. Costa VP, Kuzniec S, Molnar U, et al.: Clinical findings and haemodynamic changes associated with severe occlusive carotid artery disease. Ophthalmology 1997;104:1994-2002
  7. Ross RT, Morrow IM.: Ocular and cerebral ischémie mechanisms in disease of the internal carotid artery. Can J Neurol Sei 1984; 11:262-268
  8. Brown GC, Magargal LE.: The ocular ischémie syndrome. Clinical, fluorescein angiographie and carotid angiographie features. Int Ophthalmol 1988; 11:239-251
  9. Mizener JB, Podhajsky R Hayreh SS. Ocular ischémie syndrome. Ophthalmology 1997;TJ4*:859j:864
  10. Jacobs NA, Ridgway AE.: Syndrome of ischémie ocular inlammation:six cases and a review. Br J Ophthatmol 1985;69:681-687
  11. Svalingam A, Brown GC, Magargal LE.: The ocular ischémie syndrome. III. Visual prognosis and the effect of treatment, fnt Ophthalmol 1991;15:15-20
  12. Ros MA, Magargal LE, Hedges TR, et al.: Ocular ischémie syndrome:Long term ocular complications. Ann Ophthalmol 1987;19:270-272
  13. Michelson PE, Knox DL, Gren WR.: Ischémie ocular inflammation. Arch Ophthalmol 1971;86:274-280
  14. Murphy R, Wilson RM, Talbot JE: A case of ocular ischémie syndrome in a young insulin dependent diabetic male. Diabetes Res Clin Pract 1993; 19:245-248
  15. Kiser WD.: Recovery of vision following treatment of the ocular ischémie syndrome. Am J Ophthalmol 1983:15:305-310
  16. Casson RJ, Fleming FK, Shaikh A, et al.: Bilateral ocular ischémie syndrome secondary to giant cell arteritis. Arch Ophthalmol 2001;119:306:307
  17. Worral M, Atebara N, Meredith T, et aL: Bilateral ocular ischémie syndrome in takayasu disease. Retina 2001;21:73-74
  18. Onishi MN, Okamoto N, Suzuki A, et al.: Report of a patient with aortic dissection evolving in to binocular ischémie retinopathy. Graefe's Arch Clin Exp Ophthalmol 2000;238:200-202
  19. Barrall JL, Summers CG.: Ocular ischémie syndrome in a child Moyambya disease and neurofibromatosis. Surv Ophthalmol 1996;40:500-504.