Osteomalacia is a metabolic bone disease that results from defective bone mineralization and may present with a variety of clinical and radiographie manifestations mimicking other musculoskeletal disorders, In this article a case of osteomalacia showing the characteristics of spondyloarthropaty was presented.
📌 3 Key Takeaways
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Objective & Hypothesis:Osteomalacia is a metabolic bone disease that results from defective bone mineralization and may present with a variety of clinical and radiographie manifestations mimicking other musculoskeletal disorders, In this article a case of osteomalacia showing the characteristics of spondyloarthropaty was presented.
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Key Findings & Data:Osteomalacia is a metabolic bone disease that results from defective bone mineralization and may present with a variety of clinical and radiographie manifestations mimicking other musculoskeletal disorders, In this article a case of osteomalacia showing the characteristics of spondyloarthropaty was presented.
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Conclusion & Impact:Osteomalacia is a metabolic bone disease that results from defective bone mineralization and may present with a variety of clinical and radiographie manifestations mimicking other musculoskeletal disorders, In this article a case of osteomalacia showing the characteristics of spondyloarthropaty was presented.
Osteomalacia is a metabolic bone disease that results from defective bone mineralization and may present with a variety of clinical and radiographie manifestations mimicking other musculoskeletal disorders, In this article a case of osteomalacia showing the characteristics of spondyloarthropaty was presented.
Özet
Osteomalazi defekiif kemik mineralizasyonu sonucu oluşan bir metabolik kemik hastalığıdır, diğer muskuloskeletal bozuklukları taklit eden çeşitli klinik ve radyografik görünümlerle ortaya çıkabilir. Bu makalede spondiloartropati özelliklen gösteren bir osteomalazi olgusu sunulmuştur.
NAKİPOĞLU, G. F., KARAMERCAN, A., KARAGÖZ, A., ÖZGİRGİN, N. (2003). A case of osteomalacia showing the characteristics of spondyloartropathy. Romatizma Dergisi, Cilt: 18 - Sayı: 1, 32–36. https://ajindex.com/articles/306014
UÛurlu H. Osteomalazi, Paget HastalÝÛÝ. In, Beyazova M, Gke-Kutsal Y, (eds) Fiziksel TÝp ve Rehabilitasyon, Ankara: Gneß Kitabevi, 2000: Cilt 2: 1894-1902
Whyte MR. Sclerosing Bone Dysplasias, Axial Osteomalacia. In, Favus MJ, ed. Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism Philadelphia: Lippincott-Raven, 1998, 327-344
Burgener FA, Kormano M. Bone and Joint Disorders. New York: Thieme Stuttgart, 1997, 3-12
Reginato AJ, Falasca GF, Pappu R, McKnight B et el. Musculoskeletal manifestations of osteomalacia:Report of 26 cases and literature review. Arthritis Rheum 1999; 28: 287-304
Nelson AM, Riggs BL, Jowsey JO. Atypical axial ostomalacia. Report of four cases with two having features of ankylosing spondylitis. Arthritis Rheum 1978; 21: 715-22
Demiaux-Domenech B, Bonjour JP, Rizzoli R. Axial osteomalacia: report of a new case with selective increase in axial bone mineral density. Bone 1996 Jun 18:633-7
Burnstein MI, Lawson JP, Kattamasu SR, Ellis BI et al. The enthesopathic changes of hypophosphatemic osteomalacia in adults: radiologic findings. Am J Roentgenol 1989 Oct 153: 785-90
Moser CR, Fessel WJ. Rheumatic Manifestations of Hypophosphatemia. Arch Intern Med 1974; 134: 674-8
Szejnfeld VL, Monier-Faugere MC, Bognar BJ, Ferraz MB. Systemic osteopenia and mineralization defect in patients with ankylosing spondylitis. J Rheumatol 1997; 24: 683-8
Takuwa Y, Matsumato T, Kurukawa T, Iizuka M et al. Calcium metabolism in paravertebral ligamentous ossification. Acta Endocrinologica 1985, 109:428-32
Gerster JC, Saudan Y, Strub-Mayor F, Guggi S. Sacroiliac changes, HLA-B27 negative, in primary hyperparathyroidism and osteomalacia. Scweiz Med Wochenschr 1979; 109:1035-40.