Journals / MN Kardiyoloji / 2008 / Cilt: 15 - Sayı: 2

The development of Gullian Barre Syndrome in a case in the course of acute coronary syndrome

Bir olguda akut koroner sendrom seyrinde gelişen Gullian Barre sendromu

Pages
126–128
DOI
—
🏛️Bibliographic metadata and full text access for this article are provided via DergiPark.
DergiPark Record ↗
✨

AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

In present, we discussed a case with Gullian Barre Syndrome who had a prior myocardial infarction and underwent coronary angiography and percutaneous coronary intervention because of unstable angina.

📌 3 Key Takeaways

🎯
Objective & Hypothesis:Guillain-Barré syndrome (GBS) is an autoimmune polyradiculoneuropathy affecting the peripheral nervous system, usually triggered by an acute infectious process.
📊
Key Findings & Data:GBS is possibly an immune response to foreign antigens (such as infectious agents, immunization and surgery), but mistargeted to host nerve tissues instead (a form of antigenic mimicry).
💡
Conclusion & Impact:In present, we discussed a case with Gullian Barre Syndrome who had a prior myocardial infarction and underwent coronary angiography and percutaneous coronary intervention because of unstable angina.
🔬Design:Research Article
📍Scope:Comprehensive Sample
⚙️Variables:Akut hastalık · Orta yaşlı · Erkek

Abstract

Guillain-Barré syndrome (GBS) is an autoimmune polyradiculoneuropathy affecting the peripheral nervous system, usually triggered by an acute infectious process. GBS is possibly an immune response to foreign antigens (such as infectious agents, immunization and surgery), but mistargeted to host nerve tissues instead (a form of antigenic mimicry). Etiology of GBS may include adverse immune response occurred after myocardial infarction. In present, we discussed a case with Gullian Barre Syndrome who had a prior myocardial infarction and underwent coronary angiography and percutaneous coronary intervention because of unstable angina.

Özet

Gullian Barre Sendromu (GBS) genellikle akut infeksiyon tarafından tetiklenen ve periferik sinir sistemini etkileyen bir otoimmün poliradikulonöropatidir. GBS’de cerrahi girişim, infeksiyon ajanlarına maruziyet ya da aşılama gibi sebeplerden doğan immün uyarılma muhtemelen antijenik benzerlikten dolayı vücudun kendi sinir dokusuna da çapraz otoimmun cevap oluşturur. GBS etyolojisinde miyokard infarktüsü sonrası gelişebilecek otoimmun mekanizmalar da sorumlu tutulabilmektedir. Burada daha önce miyokard infarktüsü geçiren, kararsız anjina nedeni ile koroner anjiyografi ve perkütan koroner girişim uygulanan bir olguda gelişen GBS durumu tartışılmıştır.

🔖

Cite This Article

ŞATIROĞLU, Ö., VURAl, M., DEMİRCİ, S., TERZİ, S. (2008). The development of Gullian Barre Syndrome in a case in the course of acute coronary syndrome. MN Kardiyoloji, Cilt: 15 - Sayı: 2, 126–128. https://ajindex.com/articles/297806

📄 Full Text PDF Viewer

📄

PDF görüntüleyici hazırlanıyor...

References (1)

  1. Chun AA, McGee SR. Bedside diagnosis of coronary artery disease: a systematic review. Am J Med 2004; 117: 334-43 2.Hughes RAC, Winer JB. Guillain-Barre syndrome. In: Matthews WB, Glaser GH, eds. Recent advances in clinical neurology 4. Edinburgh: Churchill Livingstone, 1984: 19-49. 3.Ancillo P, Duarte J, Cortina JJ, Sempere AP, Claverıa LE. Guillain-Barre syndrome after acute myocardial infarction treated with anistreplase. Chest 1994; 105:1301-2. 4.Ng E, Stafford PJ. Guillain-Barre syndrome after myocardial infarction. Int J Cardiol 2003; 90:129-30. 5.Asahina M, Kuwabara S, Suzuki A, Hattori T. Autonomic dysfunction in demyelinating axonal subtypes of Guillain-Barre syndrome. Acta Neurol Scand 2002; 105:44-50 6.Dagres N, Haude M, Baumgart D, Sack S, Erbel R. Assessment of coronary morphology and flow in a patient with Guillain-Barre syndrome and ST-segment elevation. Clin Cardiol 2001; 24:260-3. 7.Iga K, Himura Y, Izumi C, Miyamoto T, Kijima K, Gen H, Konishi T. Reversible left ventricular dysfunction associated with Guillain-Barre syndrome an expression of cate-cholamine cardiotoxicity? Jpn Circ J 1995; 59:236-40. 8.Hiraga A, Nagumo K, Suzuki K, Sakakibara Y, Kojima S. A patient with Guillain-Barre syndrome and recurrent episodes of ST elevation and left ventricular hypokinesis in the anterior wall. No To Shinkei 2003; 55:517-20. 9. Bernstein R, Mayer SA, Magnano A. Neurogenic stunned myocardium in Guillain-Barre syndrome. Neurology 2000; 54:759-62. 10.Pfeiffer G, Schiller B, Kruse J, Netzer J. Indicators of dysautonomia in severe Guillain-Barre syndrome. J Neurol. 1999; 246:1015-22 11.Flachenecker P, Toyka KV, Reiners K. Cardiac arrhythmias in Guillain-Barre syndrome. An overview of the diagnosis of a rare but potentially life-threatening complication. Nervenarzt 2001; 72:610-7 12.Zöllei E, Avramov K, Gingl Z, Rudas L. Severe cardiovascular autonomic dysfunction in a patient with Guillain-Barre syndrome: a case report. Auton Neurosci 2000 28;86:94-8 13.Dressier W. A post-myocardial infarction syndrome; preliminary report of a complication resembling idiopathic, recurrent, benign pericarditis. J Am Med Assoc 1956; 160:1379-83. 14.Mcdonagh AJ, Dawson J. Guillain-Barre syndrome after myocardial infarction. Br Med J (Clin Res Ed) 1987;294: 613-4. 15.Arnason BG, Asbury AK. Idiopathic polyneuritis after surgery. Arch Neurol 1968;18:500-7.