SavedAdd your journal

Journals / Bakırköy Tıp Dergisi / 2010 / Cilt: 6 - Sayı: 1

Hipotiroidinin eşlik ettiği tekrarlayan spontan ovaryan hiperstimülasyon sendromu: Olgu sunumu

Recurrent spontaneous ovarian hyperstimulation syndrome with hypothyroidism: A case report

Ebru AKBAY
Cihangir UZUNÇAKMAK
Nadire Sevda İDİL
Zeynep AKÇIĞ
Gül ÖZEL
LEVENT YAŞAR
Pages
42–45
DOI
—
🏛️Bibliographic metadata and full text access for this article are provided via DergiPark.
DergiPark Record ↗
✨

AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

Hypothyroidism is another risk factor for the development of spontaneous ovarian hyperstimulation.

📌 3 Key Takeaways

🎯
Objective & Hypothesis:Ovarian hyperstimulation most commonly occurs iatrogenically in association with assisted reproductice technologies.
📊
Key Findings & Data:Incidence of iatrogenic ovarian hyperstimulation is 0,2-1%.
💡
Conclusion & Impact:Hypothyroidism is another risk factor for the development of spontaneous ovarian hyperstimulation.
🔬Design:Research Article
📍Scope:Comprehensive Sample
⚙️Variables:Domain Parameters

Abstract

Ovarian hyperstimulation most commonly occurs iatrogenically in association with assisted reproductice technologies. Incidence of iatrogenic ovarian hyperstimulation is 0,2-1%. There are only a few cases of pregnancies associated with recurrent spontaneous ovarian hyperstimulation. This report describes a case in which a naturally conceived pregnancy was associated with recurrent spontaneous ovarian hyperstimulation with hypothyroidism. The etiopathogenesis of spontaneous ovarian hyperstimulation is unclear. It was suggested that polycystic ovary syndrome, twin and molar pregnancies in which the endogenous hCG levels were higher than normal, mutation in the FSH receptor could be risk factors for spontaneous ovarian hyperstimulation. Hypothyroidism is another risk factor for the development of spontaneous ovarian hyperstimulation.

Özet

Ovarian hiperstimülasyon yardımcı üreme teknikleri ile birlikte gelişen iyatrojenik bir durumdur. İyatrojenik ovarian hiperstimulasyonun insidansı %0,2-1 arasynda değişmektedir. Literatürde nadir olarak spontan hiperstimülasyon sendromu bildirilmiştir. Makalede hipotiroidinin eşlik ettiği spontan rekürren ovarian hiperstimülasyon sendromu tanımlanmıştır. Spontan ovarian hiperstimülasyon sendromunun etyopatogenezi bilinmemektedir. Polikistik over sendromu, endojen hCG seviyelerinin yüksek seyrettiği çoğul gebelikler ve molar gebelikler, FSH reseptör mutasyonu varlığı ve hipotiroidi risk faktörleri olarak saptanmıştır.

🔖

Cite This Article

AKBAY, E., UZUNÇAKMAK, C., İDİL, N. S., AKÇIĞ, Z., ÖZEL, G., YAŞAR, L. (2010). Hipotiroidinin eşlik ettiği tekrarlayan spontan ovaryan hiperstimülasyon sendromu: Olgu sunumu. Bakırköy Tıp Dergisi, Cilt: 6 - Sayı: 1, 42–45. https://ajindex.com/articles/132894

📄 Full Text PDF Viewer

📄

PDF görüntüleyici hazırlanıyor...

References (13)

  1. Abu-Louz SK, Ahmed AA, Swan RW. Spontaneous ovarian hyperstimulation syndrome with pregnancy. Am J Obstet Gynecol 1997; 177: 476-477
  2. Smits G, Olatunbosun O, Delbaere A, Pierson R, Vassart G, Costagliola S. Ovarian hyperstimulation syndrome due to a mutation in the follicle-stimulating hormone receptor. N Engl J Med 2003; 349: 760-766
  3. Zalel Y, Katz Z, Caspi B, Ben-Hur H, Dgani R, Insler V. Spontaneous ovarian hyperstimulation syndrome concomitant with spontaneous pregnancy in a woman with polycystic ovarian disease. Am J Obstet Gynecol 1992; 167: 122-124
  4. Chae HD, Park EJ, Kim SH, Kim CH, Kang BM, Chang YS. Ovarian hyperstimulation syndrome complicating a spontaneous singleton pregnancy: a case report. J Assist Reprod Genet 2001; 18: 120-123
  5. Rutkowski A, Dubinsky I. Ovarian hyperstimulation syndrome: imperatives for the emergency physician. J Emerg Med 1999; 17: 669-672
  6. Abramov Y, Schenker JG, Levin A, Friedler S, Nisman B, Barak V. Plasma inflammatory cytokines correlate to the ovarian hyperstimulation syndrome. Hum Reprod 1996; 11: 1381-1386
  7. Delbaere A, Bergmann PJ, Gervy-Decoster C, et al. Increased angiotensin II in ascites during severe ovarian hyperstimulation syndrome: role of early pregnancy and ovarian gonadotropin stimulation. Fertil Steril 1997; 67: 1038-1045
  8. Abramov Y, Barak V, Nisman B, Schenker JG. Vascular endothelial growth factor plasma levels correlate to the clinical picture in severe ovarian hyperstimulation syndrome. Fertil Steril 1997; 67: 261-265
  9. Krasnow JS, Berga SL, Guzick DS, Zeleznik AJ, Yeo KT. Vascular permeability factor and vascular endothelial growth factor in ovarian hyperstimulation syndrome: a preliminary report. Fertil Steril 1996; 65: 552-555
  10. Ludwig M, Bauer O, Lopens A, Jelkmann W, Diedrich K. Serum concentrations of vascular endothelial growth factor cannot predict the course of severe ovarian hyperstimulation syndrome. Hum Reprod 1998; 13: 30-32
  11. Zalel Y, Orvieto R, Ben-Rafael Z, Homburg R, Fisher O, Insler V. Recurrent spontaneous ovarian hyperstimulation syndrome associated with polycystic ovary syndrome. Gynecol Endocrinol 1995; 9: 313-315
  12. Rosen GF, Lew MW. Severe ovarian hyperstimulation syndrome in a spontaneous singleton pregnancy. Am J Obstet Gynecol 1991; 165: 1312-1313
  13. Chae HD, Park EJ, Kim SH, Kim CH, Kang BM, Chang YS. Ovarian hyperstimulation syndrome complicating a spontaneous singleton pregnancy: a case report. J Assist Reprod Genet 2001; 18: 120-123.