Journals / Bakırköy Tıp Dergisi / 2016 / Cilt: 12 - Sayı: 4

Livedoid vaskülopatide anestezi ve düşük molekül ağırlıklı heparin kullanımı

Livedoid Vasculopaty and Anesthetic Management in Cesarean Delivery

Pages
224–226
DOI
—
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AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

This case report describes successful anesthetic management of two patients with livedoid vasculopathy.

📌 3 Key Takeaways

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Objective & Hypothesis:Livedoid vasculopathy (LV) is a hyalinizing vascular disease characterized by painful purple macules and papules that subsequently ulcerate.
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Key Findings & Data:This vasculopathy may be associated with chronic venous insufficiency, deep venous thrombosis, factor V Leiden mutation, protein C deficiency, antiphospholipid syndrome, increased homocysteine levels, abnormalities in fibrinolysis, increased platelet activation and sickle cell disease.
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Conclusion & Impact:This case report describes successful anesthetic management of two patients with livedoid vasculopathy.
🔬Design:Research Article
📍Scope:Comprehensive Sample
⚙️Variables:Domain Parameters

Abstract

Livedoid vasculopathy (LV) is a hyalinizing vascular disease characterized by painful purple macules and papules that subsequently ulcerate. This vasculopathy may be associated with chronic venous insufficiency, deep venous thrombosis, factor V Leiden mutation, protein C deficiency, antiphospholipid syndrome, increased homocysteine levels, abnormalities in fibrinolysis, increased platelet activation and sickle cell disease. Difficult venous access, unreliable measurement of peripheral O2 saturation and increased susceptibility to venous embolic events may be a challenge for anesthetists. There is limited data about anesthetic management of livedoid vasculopathy in the literature. This case report describes successful anesthetic management of two patients with livedoid vasculopathy.

Özet

Livedoid vaskülopati (LV) hyalinizan vasküler hastalık olup ağrılı mor maküller, papüller ve ulserasyonla karekterizedir. Bu vaskülopati kronik venöz yetmezlik, derin ven trombozu, Faktör V Leiden mutasyonu, protein C eksikliği, antifosfolipid sedrom, artmış homosistein seviyeleri, anormal fibrinoliz, artmış platelet aktivasyonu ve orak hücre hastalığı ile ilişkilidir. Venöz girişim güçlüğü, güvenilmez periferal O2 satürasyonu venöz embolik olaylara artmış yatkınlık anestezistin başetmesi gereken olaylardır. Literatürde LV'de anestezi ile ilgili sınırlı bilgi bulunmaktadır. Bu vaka sunumu LV'li 2 hastanın başarılı anestezi yöntemini sunmaktadır.

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Cite This Article

BAŞARANOĞLU, G., İDİN, K., UMUTOĞLU, T., ESEN, A., BAKAN, M., SALİHOĞLU, Z. (2016). Livedoid vaskülopatide anestezi ve düşük molekül ağırlıklı heparin kullanımı. Bakırköy Tıp Dergisi, Cilt: 12 - Sayı: 4, 224–226. https://ajindex.com/articles/132569

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References (5)

  1. Bartd JW, Winkelmann RK. Livedo vasculitis. Segmental hyalinizing vasculitis of the dermis. Arch Dermatol 1967; 96: 489-499.
  2. Gonzalez-Santiago TM, Davis MD. Update of management of connective tissue diseases: livedoid vasculopathy. Dermatol Ther 2012; 25: 183-194.
  3. Hairston BR, Davis MD, Pittelkow MR, Ahmed I. Livedoid vasculopathy: further evidence for procoagulant pathogenesis. Arch Dermatol 2006; 142: 1413-1418.
  4. Scavone MB, Ratliff J, Wong CA. Physiologic effects of neuraxial anesthesia. In: Spinal and epidural anesthesia. (Ed: Wong CA) The McGraw-Hill Companies; 2007: p.111-126.
  5. Abou Rahal J, Ishak RS, Otrock ZK, Kibbi AG, Taher AT. Livedoid vasculopathy in a patient with lupus anticoagulant and MTHFR mutation: treatment with low-molecular-weight heparin. J Thromb Thrombolysis 2012; 34: 541-544.