Dergiler / Cumhuriyet Dental Journal / 2015 / Cilt: 18 - Sayı: 2

Malondialdehyde levels and total antioxidant capacity in the dental follicles of the asymptomatic impacted third molars

Sayfa
108–115
Yayın Tarihi
2022-10-11
DOI
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All of tissues samples were analyzed for MDA and TAC.Results: Levels of the MDA and TAC in DFs were significantly higher than the levels of MDA and TAC provided from healthy gingival tissues of the same patients (p

📌 3 Maddede Bu Makale

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Amaç & Hipotez:Objectives: Malondialdehyde (MDA), is one of many low molecular weight end products of lipid peroxidation (LPO), increases in oxidative stress.
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Temel Bulgular & Veri:Materials and Methods: This study involved 40 DFs of 40 patients referred for clinically and radiographically asymptomatic ITMs. 40 healthy gingival tissues in the same patients obtained during surgical removal of teeth as a control group.
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Sonuç & Katkı:All of tissues samples were analyzed for MDA and TAC.Results: Levels of the MDA and TAC in DFs were significantly higher than the levels of MDA and TAC provided from healthy gingival tissues of the same patients (p
🔬Tür:Araştırma Makalesi
📍Kapsam:Genişletilmiş Akademik Örneklem
⚙️Değişkenler:Alan Parametreleri

Abstract

Objectives: Malondialdehyde (MDA), is one of many low molecular weight end products of lipid peroxidation (LPO), increases in oxidative stress. Antioxidants such as total antioxidant capacity (TAC) have a protective effect against reactive oxygen species. The aim of this study is to examine the development of the antioxidant defense mechanism in dental follicles (DFs) of radiologically asymptomatic impacted third molars (ITMs) by using MDA and TAC. Materials and Methods: This study involved 40 DFs of 40 patients referred for clinically and radiographically asymptomatic ITMs. 40 healthy gingival tissues in the same patients obtained during surgical removal of teeth as a control group. This study involved DFs widths on periapical radiographs narrower than 2.5 mm were included in the study. All of tissues samples were analyzed for MDA and TAC.Results: Levels of the MDA and TAC in DFs were significantly higher than the levels of MDA and TAC provided from healthy gingival tissues of the same patients (p

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Bu Makaleye Atıf Yap

DOGAN, O., TEKİN, U., ATİL, F., GURCAN, S., KİSA, U., KARSİYAKA, M. H. (2022). Malondialdehyde levels and total antioxidant capacity in the dental follicles of the asymptomatic impacted third molars. Cumhuriyet Dental Journal, Cilt: 18 - Sayı: 2, 108–115. https://ajindex.com/articles/477115

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Kaynakça (12)

  1. Adelsperger J, Campbell JH, Coates DB, Summerlin DJ, Tomich CE. Early soft tissue pathosis associated with impacted third molars without pericoronal radiolucency. Oral Surg Oral Med Oral Pathol Oral Radiol Endod 2000;89:402-406.
  2. Glosser JW, Campbell JH. Pathologic change in soft tissues associated with radiographically 'normal' third molar impactions. Br J Oral Maxillofac Surg 1999;37:259-260.
  3. Güven O, Keskin A, Akal UK. The incidence of cysts and tumors around impacted third molars. Int J Oral Maxillofac Surg 2000;29:131-135.
  4. Saraçoğlu U, Kurt B, Günhan O, Güven O. MIB-1 expression in odontogenic epithelial rests, epithelium of healthy oral mucosa and epithelium of selected odontogenic cysts. An immunohistochemical study. Int J Oral Maxillofac Surg 2005;34:432-435. No authors listed. NIH consensus development conference for removal of third molars. J Oral Surg 1980;38:235-236.
  5. Stanley HR, Alattar M, Collett WK, Stringfellow HR JR, Spiegel EH. Pathological sequelae of "neglected" impacted third molars. J Oral Pathol 1988;17:113-117.
  6. Stephens RG, Kogon SL, Reid JA. The unerupted or impacted third molar--a critical appraisal of its pathologic potential. J Can Dent Assoc 1989;55:201-207.
  7. Eliasson S, Heimdahl A, Nordenram A. Pathological changes related to long-term impaction of third molars. A radiographic study. Int J Oral Maxillofac Surg 1989;18:210-212.9. Sies H. Oxidative stress: oxidants and antioxidants. Exp Physiol 1997;82:291-295
  8. Chapple IL, Matthews JB. The role of reactive oxygen and antioxidant species in periodontal tissue destruction. Periodontol 2000 2007;43:160-232.
  9. Chapple IL, Mason GI, Garner ZI, GH, Maxwell SR, Whitehead TP. Enhanced chemiluminescent assay for measuring the total antioxidant capacity of serum, saliva and crevicular fluid. Ann Clin Biochem 1997;34:412-421.
  10. Halliwell B, Gutteridge JMC. Oxidative stress: adaptation, damage, repair and death. In: Halliwell B, Gutteridge JMC (eds). Free radicals in biology and medicine. Oxford, UK: Oxford University Press, 1999:284- 330. Luqman S, Rizvi SI. Protection of lipid peroxidation and carbonyl formation in proteins by capsaicin in human erythrocytes subjected to oxidative stress. Phytother Res 2006; 20:303- 306. Little RE, Gladen BC. Levels of lipid peroxides in uncomplicated pregnancy: a review of the literature. Reprod Toxicol 1999;13:347-352.
  11. Poston L, Raijmakers MT. Trophoblast oxidative stress, antioxidants and pregnancy outcome-a review. Placenta 2004;25:72-78.
  12. Akpinar A, Toker H,