Journals / Haseki Tıp Bülteni / 2022 / Cilt: 60 - Sayı: 3

Determination of the Relation Between Passive Cigarette Smoking in Children and Respiratory Tract Infections by Evaluation of Urine Cotinine/Creatinine Levels

Pages
254–262
Publication Date
2022-10-17
DOI
—
🏛️Bibliographic metadata and full text access for this article are provided via DergiPark.
DergiPark Record ↗
✨

AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

Bronchopneumonia and sinus infections were statistically significant in the case group (p

📌 3 Key Takeaways

🎯
Objective & Hypothesis:Aim: Passive smoking is an important public health issue due to the clinical problems it causes.
📊
Key Findings & Data:Methods: Seventy-two children who came to the Pediatric Outpatient Clinic at Istanbul Bagcilar Training and Research Hospital for a check-up with no current health problems between November 2020 and March 2021 were included in this prospective cross-sectional study.
💡
Conclusion & Impact:Bronchopneumonia and sinus infections were statistically significant in the case group (p
🔬Design:Survey & Empirical Study
📍Scope:2020 Verileri
⚙️Variables:Domain Parameters

Abstract

Aim: Passive smoking is an important public health issue due to the clinical problems it causes. In this study, we determined the effect of passive smoking on respiratory tract infections using a survey method, family history, and urine cotinine/creatinine ratio. Methods: Seventy-two children who came to the Pediatric Outpatient Clinic at Istanbul Bagcilar Training and Research Hospital for a check-up with no current health problems between November 2020 and March 2021 were included in this prospective cross-sectional study. The study group included 36 children with at least one active smoker in the house, and the control group included 36 children with no active smokers in the house. With the survey, sociodemographic variables about the family and child as well as the frequency of lower or upper respiratory tract infection history were questioned. Cotinine and creatinine levels were measured using the urine samples of the patients included in the study. Results: The frequency of respiratory tract infections in the last two years was increased in the group with an active smoker in the household. The frequency of bronchopneumonia in the case group was 44.4%, whereas it was 5.6% in the control group. The sinus infection was seen in 22.2% of those in the case group, while there were no sinus infections reported in the control group. Bronchopneumonia and sinus infections were statistically significant in the case group (p

🔖

Cite This Article

IŞIKLI, S. M., ÖZEL, A., EROL, M., GAYRET, Ö. B., BUKE, Ö., ASLAN, S. (2022). Determination of the Relation Between Passive Cigarette Smoking in Children and Respiratory Tract Infections by Evaluation of Urine Cotinine/Creatinine Levels. Haseki Tıp Bülteni, Cilt: 60 - Sayı: 3, 254–262. https://ajindex.com/articles/534964

📄 Full Text PDF Viewer

📄

PDF görüntüleyici hazırlanıyor...

References (16)

  1. World Health Organisation. Tobacco Atlas 10. Erişim: http//:www.who.int/tobaco/en/atlas/10.pdf
  2. Vineis P, Hoek G, Krzyzanowski M, et al. Lung cancers attributable to environmental tobacco smoke and air pollution in non-smokers in different European countries: a prospective study. Environ Health 2007;6:7
  3. Boyacı H, Duman C, Basyigit I, Ilgazlı A, Yıldız F. lkokul çocuklarında çevresel sigara dumanına maruziyetin idrar kotinin düzeyi ile degerlendirilmesi. Tuberk Toraks 2004;52:231-6
  4. Keskinoglu P, Aksakoglu G. Pasif sigara içiciliginin solunum sistemi üzerine etkileri. Türk Ped Ars 2007;42:136-41
  5. Harlap S, Davies AM. Infant admissions to hospital and maternal smoking. Lancet 1974;1:529-32
  6. Neri M, Ugolini D, Bonassi S, et al. Children’s exposure to environmental pollutants and biomarkers of genetic damage. II. Results of a comprehensive literature search and metaanalysis. Mutat Res 2006;612:14-39
  7. Matt GE, Wahlgren DR, Hovell MF, et al. Measuring environmental tobacco smoke exposure in infants and young children through urine cotinine and memory-based parental reports: empirical findings and discussion. Tob Control 1999;8:282-9
  8. Zhou S, Rosenthal DG, Sherman S, Zelikoff J, Gordon T, Weitzman M. Physical, behavioral, and cognitive effects of prenatal tobacco and postnatal secondhand smoke exposure. Curr Probl Pediatr Adolesc Health Care 2014;44:219-41
  9. Bülbül HS, Ceyhun G. Pasif sigara içiciligi. Aile Hekimligi Dergisi 2006;10:123-8
  10. Taioli E. Gene-environment interaction in tobacco-related cancers. Carcinogenesis 2008;29:1467-74
  11. Boztas G, Aslan D, Bilir N. Çevresel Sigara Dumanından Etkilenim ve Çocuklar. STED 2006;5:75-8
  12. Kramer MS. Intrauterine growth and gestational duration determinants. Pediatrics 1987;80:502-11
  13. Gursoy ST, Soyer MT, Ocek Z, Ciceklioglu M, Aksu F. Why are Turkish children at risk of exposure to environmental tobacco smoke in their homes? Asian Pac J Cancer Prev 2008;9:467- 72
  14. Zafar Ullah AN, Huque R, Akter S, et al. Children’s exposure to second-hand smoke at home in Bangladesh: a community survey. BMJ Open 2013;3:e003059
  15. Liao YM, Chen YT, Kuo LC, Chen PL. Factors associated with parental smoking in the presence of school-aged children: a cross-sectional study. BMC Public Health 2013;13:819
  16. Wang CP, Ma SJ, Xu XF, Wang JF, Mei CZ, Yang GH. The prevalence of household second-hand smoke exposure and its correlated factors in six counties of China. Tob Control 2009;18:121-6