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Journals / Çukurova Üniversitesi Tıp Fakültesi Dergisi / 1998 / Cilt: 23 - Sayı: 4

Hemiballism-hemichorea: Report of 28 cases

Hemiballismus-Hemikore: 28 vaka analizi

Sibel KARŞIDAĞ
Feriha ÖZER
Mustafa UFACIK
Sibel ÇETİN
Sibel KIZGIN
Şebnem ALTINLI
Baki ARPACI
Pages
202–206
Publication Date
2022-10-10
DOI
—
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AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

In 2 patients who underwent CT alone, a responsible lesion was not identified.

📌 3 Key Takeaways

🎯
Objective & Hypothesis:We compared cranial CT or magnetic resonance imaging in twenty-eight patients with hemiballism-hemichorea secondary to vascular brain damage.
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Key Findings & Data:Causes of hemiballismus-hemikore included infarction (15 patients), hemorrhage (7 patients), hyperglycemia (3 patients), and hypoglycemia (1 patient).
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Conclusion & Impact:In 2 patients who underwent CT alone, a responsible lesion was not identified.
🔬Design:Research Article
📍Scope:Comprehensive Sample
⚙️Variables:Domain Parameters

Abstract

We compared cranial CT or magnetic resonance imaging in twenty-eight patients with hemiballism-hemichorea secondary to vascular brain damage. Sites considered as possible locations for a lesion causing hemiballismus-hemikore included the contralateral putamen, thalamus, caudate nucleus, subthalamic nucleus, pallidum, striatum, premotor-motor cortex, corona radiata, and ipsilateral thalamus. Causes of hemiballismus-hemikore included infarction (15 patients), hemorrhage (7 patients), hyperglycemia (3 patients), and hypoglycemia (1 patient). In 2 patients who underwent CT alone, a responsible lesion was not identified.

Özet

Bu çalışmada vasküler beyin hasarına bağlı olarak gelişen 28 hemiballismus-hemikore hastasında kranial CT veya manyetik rezonans görüntüleme bulgularını karşılaştırdık. Hemiballismusa-hemikoreye neden olan lezyonlar, kontrlateral putamen, talamus, kaudat nukleus, subtalamik nukleus, pallidum, striatum, premotor-motor korteks, korona radiata ve ipsilateral talamusu kapsamakta idi. Hemiballismus-hemikore sebebleri 15 hastada enfarkt, 7 hastada hemoraji, 3 hastada hiperglisemi, 1 hastada hipoglisemiye bağlandı. Sadece CT yapılan 2 hastada ise uyumlu lezyon saptanamadı.

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Cite This Article

KARŞIDAĞ, S., ÖZER, F., UFACIK, M., ÇETİN, S., KIZGIN, S., ALTINLI, Ş., ARPACI, B. (2022). Hemiballism-hemichorea: Report of 28 cases. Çukurova Üniversitesi Tıp Fakültesi Dergisi, Cilt: 23 - Sayı: 4, 202–206. https://ajindex.com/articles/486264

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References (18)

  1. Altafullah I, Pascual-Leone A, Duvall K,et al. Putaminal hemorrhage accompanied by hemichorea-hemiballism. Stroke 1990; 21: 1093- 1094
  2. Berardelli A, Stocchi F, Gallucci M et al: Neurophysiological and magnetic resonance studies in a patient with hemiballismus. Funct Neurol 1987; 2(1): 93-98
  3. Borgohain R, Singh AK, Thadani R et al: Hemiballismus due to an ipsilateral striatal haemorrhage: an unusual localization. J Neurol Sci 1995; 130(1): 22-24
  4. Carpay HA, Arts WF, Kloet A,et al. Hemichorea reversible after operation in a boy with cavernous angioma in the head of the caudate nucleus. J Neurol Neurosurg Psychiatry 1994; 57: 1547-1548
  5. Defebvre L, Destee A, Cassim F,et al. Transient hemiballism and striatal infarction. Stroke 1990; 21: 967-968
  6. Freilich RJ, Chambers BR. Choreoathetosis and thalamic haemorrhage. Clin Exp Neurol 1988; 25: 115-120
  7. Folstein S, Abbott M, Moses R,et al. A phenocopy of Huntington’s disease: Lacunar infarcts of the corpus striatum. Johns Hopkins Med J 1981; 148: 104-113
  8. Fukui T, Hasegawa Y, Seriyama S,et al. Hemiballism-hemichorea induced by subcortical ischemia. Can J Neurol Sci 1993; 20:324-328
  9. Garretto NS, Bueri JA, Kremenchutzky M et al: Hemichorea associated with cerebral toxoplasmosis and AIDS. Arch Neuropsiquiatr 1995; 53(1): 118- 122
  10. Giroud M, Semama D, Pradeaux L et al: Hemiballismus revealing multiple sclerosis in an infant. Childs Nerv Syst 1990; 6(4):236-238
  11. Haan J, Kremer HPH, Padberg G. Paroxismal choreoathetosis as presenting symptom of diabetes mellitus. J Neurol Neurosurg Psychiatry1989; 52: 133
  12. Hefter H, Mayer P, Benecke R. Persistent chorea after recurrent hypoglycemia. Eur Neurol 1993; 33: 244-247
  13. Kase CS, Maulsby GO, deJuan EE, et al. Hemichorea-hemiballism and lacunar infarction of the basal ganglia. Neurology 1981; 31: 452-455
  14. Katsumata T, Kitamura S, Inamura K et al: Clinical and CT findings in hemiballismus. Nippon Ronen Igakkai Zasshi 1992; 29(2): 123-128
  15. Kimura M: Physiological bases of involuntary movement. Rinsho Shinkeigaku 1994; 34(12): 1241-1242
  16. Kirk A, Harding SR. Cardioembolic caudate infarction as a cause of hemichorea in lupus anticoagulant syndrome. Can J Neurol Sci 1993;20: 162-164
  17. Lin JJ, Chang MK. Hemiballism-hemichorea and non-ketotic hyperglycemia. J Neurol Neurosurg Psychiatry 1994; 57: 748-750
  18. Linazasoro G, Urtasun M, Poza JJ, et al. Generalized chorea induced by by nonketotic hyperglycemia. Mov Disord 1993; 8: 119