We report a case with transient LSFB in the scenario of ST-segment elevation myocardial infarction (STEMI) because of acute occlusion of LAD.
📌 3 Key Takeaways
🎯
Objective & Hypothesis:Although the conduction abnormalities of the left anterior and posterior fascicles, left anterior fascicular block (LAFB), and left posterior fascicular block (LPFB) are commonly diagnosed and well-known to physicians, the abnormalities of the left septal fascicle (LSF) and left septal fascicular block (LSFB), are easily overlooked because most physicians are not aware of the existence of the LSF.
📊
Key Findings & Data:LSFB exclusively affects precordial leads without modifying the ECG in the frontal plane, causing prominent anterior QRS forces.
💡
Conclusion & Impact:We report a case with transient LSFB in the scenario of ST-segment elevation myocardial infarction (STEMI) because of acute occlusion of LAD.
🔬Design:Research Article
📍Scope:Comprehensive Sample
⚙️Variables:Domain Parameters
Özet
Although the conduction abnormalities of the left anterior and posterior fascicles, left anterior fascicular block (LAFB), and left posterior fascicular block (LPFB) are commonly diagnosed and well-known to physicians, the abnormalities of the left septal fascicle (LSF) and left septal fascicular block (LSFB), are easily overlooked because most physicians are not aware of the existence of the LSF. LSFB exclusively affects precordial leads without modifying the ECG in the frontal plane, causing prominent anterior QRS forces. LSF is irrigated exclusively by the left anterior descending (LAD) coronary artery, and therefore, LAD occlusion can result in LSFB. We report a case with transient LSFB in the scenario of ST-segment elevation myocardial infarction (STEMI) because of acute occlusion of LAD.
🔖
Cite This Article
YU, S., CHEN, H., Lİ, H. (2021). Transient left septal fascicular block in the scenario of ST-segment elevation myocardial infarction. The Anatolian Journal of Cardiology, Cilt: 25 - Sayı: 8, 588–592. https://ajindex.com/articles/403145
Cullen EL, Breen JF, Rihal CS, Simari RD, Ammash NM. Levoatriocardinal vein with partial anomalous venous return and a bidirectional shunt. Circulation 2012
e174-7. [Crossref] 2. Bernstein HS, Moore P, Stanger P, Silverman NH. The levoatriocardinal vein: morphology and echocardiographic identification of the pulmonary-systemic connection. J Am Coll Cardiol 1995
[Crossref] 3. Amoretti F, Cerillo AG, Chiappino D. The levoatriocardinal vein. Pediatr Cardiol 2005
[Crossref] 4. Maemura S, Ishizuka M, Nakata R, Motozawa Y, Yamamoto K, Takizawa M, et al. Pulmonary hypertension caused by persistent anomalous vertical vein bridging the left subclavian vein and left atrium with hypertrophic cardiomyopathy. Circulation 2014
e153-6. [Crossref] 5. Melacini P, Basso C, Angelini A, Calore C, Bobbo F, Tokajuk B, et al. Clinicopathological profiles of progressive heart failure in hypertrophic cardiomyopathy. Eur Heart J 2010
[Crossref] 6. Tweddell JS. The vertical vein: to ligate or not to ligate. J Thorac Cardiovasc Surg 2007
[Crossref] 7. Cope JT, Banks D, McDaniel NL, Shockey KS, Nolan SP, Kron IL. Is vertical vein ligation necessary in repair of total anomalous pulmonary venous connection? Ann Thorac Surg 1997
[Crossref] 8. Ong KC, Geske JB, Hebl VB, Nishimura RA, Schaff HV, Ackerman MJ, et al. Pulmonary hypertension is associated with worse survival in hypertrophic cardiomyopathy. Eur Heart J Cardiovasc Imaging 2016
[Crossref] 9. Ural E. Septal ablation. Turk Kardiyol Dern Ars 2018