Severe hypocalcemia and hypercalciuria due to contrast medium in the course of acute myocardial infarction

References (6)

1. Claeys MJ, de Meester A, Convens C, Dubois P, Boland J, De Raedt H, et al. Contemporary mortality differences between primary percutaneous coronary intervention and thrombolysis in ST-segment elevation myocardial infarction. Arch Intern Med 2011; 171: 544-9
2. Habeb M, Ağaç MT, Aliyev F, Pehlivanoğlu S, Öngen Z. Contrast media induced nephropathy: clinical burden and current attempts for prevention. Anadolu Kardiyol Derg 2005; 5: 124-9
3. Bolognese L, Falsini G, Schwenke C, Grotti S, Limbruno U, Liistro F, et al. Impact of iso-osmolar versus low-osmolar contrast agents on contrast induced nephropathy and tissue reperfusion in unselected patients with ST-segment elevation myocardial infarction undergoing primary percutaneous coronary intervention [from the Contrast Media and Nephrotoxicity Following Primary Angioplasty for Acute Myocardial Infarction (CONTRAST-AMI) Trial]. Am J Cardiol 2012; 109: 67-74
4. Blaine J, Chonchol M, Levi M. Renal control of calcium, phosphate and magnesium homeostasis. Clin J Am Soc Nephrol 2015; 10: 1257-72
5. Ludwig U, Connemann J, Keller F. Effect of low-osmolar contrast medium iopromide and iso-osmolar iodixanol on DNA fragmentation in renal tubular cell culture. Clin Exp Nephrol 2013; 17: 779-82
6. Zhao J, Huang Y, Song Y, Zhao X, Jin J, Wang J, et al. Low osmolar contrast medium induces cellular injury and disruption of calcium homeostasis in rat glomerular endothelial cells in vitro. Toxicol Lett 2009; 185: 124-31.