Journals / MN Kardiyoloji / 2000 / Cilt: 7 - Sayı: 5

Heart rate variability in congestive heart failure due to dilated cardiomyopathy

Dilate kardiyomiyopati nedeni ile oluşan konjestif kalp yetmezliğinde kalp hızı değişkenliği

Pages
370–374
DOI
—
🏛️Bibliographic metadata and full text access for this article are provided via DergiPark.
DergiPark Record ↗
✨

AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

CONCLUSIONS: Decreased heart rate variability in patients with congestive heart failure caused by dilated cardiomyopathy is related to left ventricular systolic function, ventricular rhythm problems and cardiac death, so it is important for evaluation of prognosis.

📌 3 Key Takeaways

🎯
Objective & Hypothesis:AIM: This study was designed to evaluate heart rate variability in patients with congestive heart failure caused by dilated cardiomyopathy, to determine its correlation with left ventricular systolic function and ventricular arrhythmias and to evaluate its prognostic value.
📊
Key Findings & Data:MATERIAL AND METHODS: Time domain analysis of heart rate variability on 24 hour electrocardiographic recording was performed in 60 patients with congestive heart failure (16 female, 52.7±17.3 years old) and results were compared with those in 60 control subjects (15 female, 51.6±13.8 years old, p>0.05).
💡
Conclusion & Impact:CONCLUSIONS: Decreased heart rate variability in patients with congestive heart failure caused by dilated cardiomyopathy is related to left ventricular systolic function, ventricular rhythm problems and cardiac death, so it is important for evaluation of prognosis.
🔬Design:Research Article
📍Scope:Comprehensive Sample
⚙️Variables:Ventriküler fonksiyon · sol · Kardiyomiyopati

Abstract

AIM: This study was designed to evaluate heart rate variability in patients with congestive heart failure caused by dilated cardiomyopathy, to determine its correlation with left ventricular systolic function and ventricular arrhythmias and to evaluate its prognostic value. MATERIAL AND METHODS: Time domain analysis of heart rate variability on 24 hour electrocardiographic recording was performed in 60 patients with congestive heart failure (16 female, 52.7±17.3 years old) and results were compared with those in 60 control subjects (15 female, 51.6±13.8 years old, p>0.05). RESUL TS: Patients with congestive heart failure had significantly lower values for heart rate variability than those of control subjects: RRmean (msec) (710.1±97.8 versus 827.2±149.8, p=0.01}, SDNN (msec) (86.5±32.7 versus 132.2±42.3, p=0.005), PNN 50 (%) (8.1±5.2 versus 18.1±14.7, p=0.005), rMSSD (msec) (38.0 ± 16.3 versus 68.0±44.8, p=0.005). Heart rate variability values showed negative relation with the degree of ventricular rhythm problems and occurance of death, but positive relation with the systolic functions of the left ventricle. Cardiac death showed negative relation with SDNN (r= -0.36, p=0.002), PNN50 (r= - 0.25, p=0.02), rMSSD (r= -0.27, p= 0.01), RR (r=-0.39, p=0.001), left ventricular ejection fraction (r=-0.34, p=0.003), fractional shortening (r= -0.35, p=0.003) and positive relation with the degree of ventricular arhythmias (r=039, p=0.001). CONCLUSIONS: Decreased heart rate variability in patients with congestive heart failure caused by dilated cardiomyopathy is related to left ventricular systolic function, ventricular rhythm problems and cardiac death, so it is important for evaluation of prognosis.

Özet

AMAÇ: Bu çalışma dilate kardiyomiyopati sonucu konjestif kalp yetmezliği gelişmiş olgularda kalp hızı değişkenliği parametrelerini değerlendirip, bunların sol ventrikül sistolik fonksiyonları, ventriküler aritmiler ve kardiyak ölüm gelişimi ile ilişkisini incelemek amacı ile yapılmıştır. GEREÇ VE YÖNTEM: Konjestif kalp yetmezliği bulunan 60 olgunun (14 kadın, 52.7±17.3 yaşında) 24 saatlik holter elektrokardiyografi kayıtları alındı ve sonuçları tamamen sağlıklı 60 kontrol olgusunun (15 kadın, 51.6±13.8 yaşında, p>0.05) sonuçları ile karşılaştırıldı. BULGULAR: Hasta grubunun kalp hızı değişkenliği değerleri kontrol grubuna göre belirgin olarak düşüktü; RR ortalama (msn) (710.1±47.8'e 827.2±149.8, p=0.01), SDNN (msn) (86.5±32.7'e 132.2±42.3, p=0.005), PNN50 (%) (8.1±5.2'e 18.1±14.7, p=0.005), rMSSD (msn) (38.0±16.3 'e 68.6±44.8, p=0.005). Kalp hızı değişkenliği değerleri ventriküler ritm problemlerinin derecesi ve kardiyak ölüm gelişimi ile negatif, sol ventrikül sistolik fonksiyonları ile pozitif ilişki gösteriyordu. Kardiyak ölüm SDNN (r= -0.36, p=0.002), PNN50 (r= -0.25, p=0.02), Rmssd (r= -0.27, P=0.01), RR (r= -0.39, p= 0.001), sol ventrikül ejeksiyon fraksiyonu (r= -0.34, p=0.003, fraksiyonel kısalması (r= -0.35, p=0.003) negatif, ventriküler ritm problemlerinin derecesi ile pozitif ilişki gösteriyordu (r=0.39,p=0.001). SONUÇ: Dilate kardiyomiyopati nedeni ile oluşan konjestif kalp yetmezlikli olgularda düşük kalp hızı değişkenliği parametreleri sol ventrikül sistolik fonksiyonları, ventriküler ritm problemleri ve kardiyak ölüm ile anlamlı ilişki gösterir, değerlendirilmesi prognoz açısından önem taşır.

🔖

Cite This Article

KAFTAN, H. A., KAFTAN, O. (2000). Heart rate variability in congestive heart failure due to dilated cardiomyopathy. MN Kardiyoloji, Cilt: 7 - Sayı: 5, 370–374. https://ajindex.com/articles/298094

References (1)

  1. Akselrod S, Gordon D, Libel FA, Shannon DC, Barger AC, Cohen RJ. Power spectrum analysis of heart rate fluctuation: a quantitative probe of beat to beat cardiovascular control. Science 1981; 213:220. Kleiger RE, Miller JP, Bigger JT Jr, Moss AJ, and Multicenter postinfarction Research Group. Decreased heart rate variability and its association with increased mortality after acute myocardial infarction. Am J Cardiol 1987; 59:256. Bigger JT Jr, Fleiss JL, Steinman RC, Rolnitzky LM, Kleiger RE, Rottman JN. Frequency domain measures of heart period variability and mortality after myocardial infarction. Circulation 1992; 85:164. Pomeranz B, Macaulay RJB, Caudill MA, Kutz J, Adam D, Gordon D, Kilborn KM, Barger AC, Shannon DC, Cohen RJ, Bensen H. Assessment of autonomic function in humans by heart rate spectral analysis. Am J Physiol 1985; 248: 151. Pagani M, Lombardi F, Guzzetti S, Rimoldi O, Furlan R, Pizzinelli P, Sandrone G, Malfatto G, Dell'Orto S, Piccaluga E, Turiel M, Basselli G, Cerrutti S, Malliani A. Power spectral analysis of heart rate and arterial pressure variabilities as a marker of sympathovagal interaction in man and conscious dog. Circ Res 1986; 59:178. Shannon DC, Carley DW, Benson H. Aging of modulation of heart rate. Am J Physiol 1987; 253:874. Malpas SC, Purdie GL. Circadian variation of heart rate variability. Cardiovasc Res 1990; 24:210. Saul JP, Arai Y, Berger RD, Lilly LS, Colucci WS, Cohen RJ. Assessment of autonomic regulation in chronic congestive heart failure by heart rate spectral analysis.Am J Cardiol 1988; 61:1292. Airaksinen KE, Ikaheimo MJ, Linnaluoto MK, Niemela M, Takkunen JT. Impaired vagal heart rate control in coronary artery disease. Br Heart J 1987; 58:592. Ewing DJ, Borsey DQ, Bellavere F, Clarke BF. Cardiac autonomic neuropathy in diabetes: Comparison of measures of RR interval variation. Diabetologia 1981; 21:18. Counihan PJ, Fei L, Bashir Y, Farrel TG, Haywood GA, McKenna WJ. Assessment of heart rate variability -in hypertrophic cardiomyopathy: association with clinical and prognostic features. Circulation 1993; 88;1682. Ajiki K, Murakawa Y, Yanagisawa-Miwa A, Usui M, Yamashita T, Oikawa N, Inoue H. Autonomic nervous system activity in idiopathic dilated cardiomyopathy and in- hypertrophic cardiomyopathy. Am J Cardiol 1993; 71:1316. Van Ravenswaij-Ahs CMÂ, Kolle LAA, Hpman JCW, Stoelinga GBA, Van Geijin HP. Heart rate variability. Ann Intern Med 1993; 118: 436. Malik M, Camm AJ. Heart rate variability: from facts