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Journals / Neurological sciences and neurophysiology (Online) / 2020 / Cilt: 37 - Sayı: 4

Neuroinflammation in Alzheimer’s Disease Continuum

Aslıhan TAŞKIRAN SAĞ
Müge YEMİŞÇİ
Pages
155–163
DOI
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AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

Novel findings based on advanced biotechnology and bioinformatics are covered briefly, as well.

📌 3 Key Takeaways

🎯
Objective & Hypothesis:Aging population brings an ever-increasing global burden of dementia, and Alzheimer’s disease (AD) is the most frequent type worldwide.
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Key Findings & Data:Many years of research have introduced characteristic cerebral histopathological and molecular changes in this disease.
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Conclusion & Impact:Novel findings based on advanced biotechnology and bioinformatics are covered briefly, as well.
🔬Design:Review & Systematic Survey
📍Scope:Comprehensive Sample
⚙️Variables:Domain Parameters

Özet

Aging population brings an ever-increasing global burden of dementia, and Alzheimer’s disease (AD) is the most frequent type worldwide. Many years of research have introduced characteristic cerebral histopathological and molecular changes in this disease. However, all attempts to establish an effective treatment have failed. In this review, we aim to address the basic evidence regarding the role of inflammatory mediators in AD and their link to the other pathogenetic pathways. Novel findings based on advanced biotechnology and bioinformatics are covered briefly, as well.

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Cite This Article

SAĞ, A. T., YEMİŞÇİ, M. (2020). Neuroinflammation in Alzheimer’s Disease Continuum. Neurological sciences and neurophysiology (Online), Cilt: 37 - Sayı: 4, 155–163. https://ajindex.com/articles/286251

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