Histopathological examination of the vascular structure and cataract revealed fi brin formation, reactive lens epithelial hyperplasia and vascular endothelium, which were though to be remnants of tunica vasculosa lentis.
📌 3 Key Takeaways
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Objective & Hypothesis:A 6-month-old baby was referred to our clinic for visually signifi cant cataract in the left eye.
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Key Findings & Data:Dens posterior lens opacity with hemorrhage was evident in the anterior segment evaluation.
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Conclusion & Impact:Histopathological examination of the vascular structure and cataract revealed fi brin formation, reactive lens epithelial hyperplasia and vascular endothelium, which were though to be remnants of tunica vasculosa lentis.
🔬Design:Research Article
📍Scope:Comprehensive Sample
⚙️Variables:Domain Parameters
Özet
A 6-month-old baby was referred to our clinic for visually signifi cant cataract in the left eye. Dens posterior lens opacity with hemorrhage was evident in the anterior segment evaluation. B-scan ultrasonography of the eye showed anechoic posterior segment. During the cataract surgery vascular remnants became evident after irrigation of the cloth in the anterior chamber. Histopathological examination of the vascular structure and cataract revealed fi brin formation, reactive lens epithelial hyperplasia and vascular endothelium, which were though to be remnants of tunica vasculosa lentis.
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Cite This Article
CERAN, B. B., ARIFOGLU, H., VURAL, G., TASINDI, E. (2020). Where Did That Blood Come From? Atypical Presentation of Congenital Cataract With Hemorrhage. Glokom Katarakt, Cilt: 15 - Sayı: 3, 194–196. https://ajindex.com/articles/158876
References (8)
Pau H. Hypothesis on the pathogenesis of retinopathy of prematurity – it is not VEGF alone but anatomical structures that are crucial. Graefes Arch Clin Exp Ophthalmol. 2010;248:1–3
Lambert SR, et al. Congenital Fibrovascular Pupillary Membranes: Clinical and Histopathologic Findings. Ophthalmology 2012;119:634-64 3. Cero’n O, Lou PL, Kroll AJ, et al. The vitreo-retinal manifestations of persistent hyperplasic primary vitreous (PHPV) and their management. Int Ophthalmol Clin, 2008 48:53–62 (Review
Goldberg, MF. Persistent Fetal Vasculature (PFV): An Integrated Interpretation of Signs and Symptoms Associated with Persistent Hyperplastic Primary Vitreous (PHPV) LIV Edward Jackson Memorial Lecture. Am J Ophthal 1997;124:587-626
Saint-Geniez M, D’Amore P. Development and Pathology of the Hyaloid, Choroidal, and Retinal Vasculature. Int J Dev Biol 2004;48:1045-58
Shastry BS. Persistent hyperplastic primary vitreous: congenital malformation of the eye. Clin Experiment Ophthalmol. 2009; 37:884–90
Hunt A, Rowe N, Lam A, et al. Outcomes in persistent hyperplastic primary vitreous. Br J Ophthalmol 2005;89:859- 63
Sisk RA, Berrocal AM, Feuer WJ, et al. Visual and anatomic outcomes with or without surgery in persistent fetal vasculature. Ophthalmology. 2010;117:2178-83.e1-2
Anteby I, Cohen E, Karshai I, et al. Unilateral persistent hyperplastic primary vitreous: course and outcome. J AAPOS. 2002;6:92-9 10. Fortes Filho JB, Tartarella MB, Takahagi RU, et al. Persistent fetal vasculature: ocular features, management of cataract and outcomes. Arquivos Brasileiros de Oftalmologia. 2013 19;76.