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Journals / Journal of Emergency Medicine Case Reports / 2013 / Cilt: 4 Sayı: 4

Two Consecutive Acute Coronary Syndrome Patients after Dinitro-Ortho-Cresol Exposure

Dinitro-Ortho-Cresol Maruziyeti Sonrasında Akut Koroner Sendrom Gelişen Ardışık İki Hasta

Ahmet Oğuz Baktır(Clinic of Cardiology, Kayseri Education and Research Hospital, Kayseri, Turkey)
Hayrettin Sağlam(Clinic of Cardiology, Kayseri Education and Research Hospital, Kayseri, Turkey)
Bahadır Şarlı(Clinic of Cardiology, Kayseri Education and Research Hospital, Kayseri, Turkey)
Hüseyin Arınç(Clinic of Cardiology, Kayseri Education and Research Hospital, Kayseri, Turkey)
Yasemin Doğan(Clinic of Cardiology, Kayseri Education and Research Hospital, Kayseri, Turkey)
Hüseyin Katlandur(Clinic of Cardiology, Kayseri Education and Research Hospital, Kayseri, Turkey)
Pages
147–150
Publication Date
2013-10-01
DOI
—
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AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

Further examination revealed that toxic exposure led to simultaneous acute coronary syndrome

📌 3 Key Takeaways

🎯
Objective & Hypothesis:Nitrophenols are common components of industrial effluents and have been detected in urban and agricultural waste.
📊
Key Findings & Data:The main toxic effects of DNOC derivatives are due to an increase in cellular metabolism.
💡
Conclusion & Impact:Further examination revealed that toxic exposure led to simultaneous acute coronary syndrome
🔬Design:Makale
📍Scope:Comprehensive Sample
⚙️Variables:4-Dinitrofenol · akut koroner sendrom · 4-Dinitrofenol toksik maruziyeti

Abstract

Nitrophenols are common components of industrial effluents and have been detected in urban and agricultural waste. Dinitroortho-cresol (DNOC) can easily enter the body through inhalation, ingestion or the skin. Yellow staining is typical in toxications via skin contact. The main toxic effects of DNOC derivatives are due to an increase in cellular metabolism. It acts directly on metabolism by inducing cellular oxidation and inhibiting phosphorylation, leading to the uncoupling of oxidative phosphorylation. Cardiovascular effects appear to be secondary to cellular anoxia. We report here two consecutive patients admitted to the emergency department with an initial diagnosis of toxic exposure to DNOC. Further examination revealed that toxic exposure led to simultaneous acute coronary syndrome

Özet

Nitrophenols are common components of industrial effluents and have been detected in urban and agricultural waste. Dinitro-ortho-cresol (DNOC) can easily enter the body through inhalation, ingestion or the skin. Yellow staining is typical in toxications via skin contact. The main toxic effects of DNOC derivatives are due to an increase in cellular metabolism. It acts directly on metabolism by inducing cellular oxidation and inhibiting phosphorylation, leading to the uncoupling of oxidative phosphorylation. Cardiovascular effects appear to be secondary to cellular anoxia. We report here two consecutive patients admitted to the emergency department with an initial diagnosis of toxic exposure to DNOC. Further examination revealed that toxic exposure led to simultaneous acute coronary syndrome.

🔖

Cite This Article

Baktır, A. O., Sağlam, H., Şarlı, B., Arınç, H., Doğan, Y., Katlandur, H. (2013). Two Consecutive Acute Coronary Syndrome Patients after Dinitro-Ortho-Cresol Exposure. Journal of Emergency Medicine Case Reports, Cilt: 4 Sayı: 4, 147–150. https://ajindex.com/articles/1346050

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