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AI InsightNeural therapy may be an alternative option that can be successfully applied in the treatment of these patients.
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Özet
Notalgia paresthetica (NP) is a common sensory neuropathic syndrome with the symptoms of localized pruritus and dysesthesia of the unilateral infra scapular region. NP is a rarely reported T2-T6 sensory neuropathy whose etiology and treatment are not fully established and the clinical course consists of remissions and relapses. We report two cases whom admitted to our clinic with a back burning sensation and we detected a hyperpigmented pruritic skin lesion in the scapular region. On inspection, hyperpigmented macular cutaneous lesions were seen on the scapular region. Neurological examination and biochemical blood tests were normal. Degenerative changes were observed on cervical and thoracic plain radiographs. The presence of hyperpigmented macular lesions on the scapular area of the patient and analgesic-resistant dorsal pain led us to the diagnosis of NP. We applied to these patients T2-T10 local segmental and to the hyperpigmented area with 1% lidocaine for treatment. After 2 week, the pain which was detected by a visual analog scale (VAS) declined. We applied the Leeds assessment of neuropathic symptoms and signs (LANSS) Pain Scale to analyse of of sensory dysfunction. The aim of study is to determine the efficacy of treatment of notalgia paresthetica using neural therapy , assessing the degree of improvement in itching and burning sensation. Moreover, to evaluate the signs and symptoms associated with notalgia paresthetica. As a result, NP is a disorder that should be kept in mind in the presence of neuropathic upper back pain and hyperpigmented skin lesions. Neural therapy may be an alternative option that can be successfully applied in the treatment of these patients.
Abstract
Notalgia paresthetica (NP) is a common sensory neuropathic syndrome with the symptoms of localized pruritus and dysesthesia of the unilateral infra scapular region. NP is a rarely reported T2-T6 sensory neuropathy whose etiology and treatment are not fully established and the clinical course consists of remissions and relapses. We report two cases whom admitted to our clinic with a back burning sensation and we detected a hyperpigmented pruritic skin lesion in the scapular region. On inspection, hyperpigmented macular cutaneous lesions were seen on the scapular region. Neurological examination and biochemical blood tests were normal. Degenerative changes were observed on cervical and thoracic plain radiographs. The presence of hyperpigmented macular lesions on the scapular area of the patient and analgesic-resistant dorsal pain led us to the diagnosis of NP. We applied to these patients T2-T10 local segmental and to the hyperpigmented area with 1% lidocaine for treatment. After 2 week, the pain which was detected by a visual analog scale (VAS) declined. We applied the Leeds assessment of neuropathic symptoms and signs (LANSS) Pain Scale to analyse of of sensory dysfunction. The aim of study is to determine the efficacy of treatment of notalgia paresthetica using neural therapy , assessing the degree of improvement in itching and burning sensation. Moreover, to evaluate the signs and symptoms associated with notalgia paresthetica. As a result, NP is a disorder that should be kept in mind in the presence of neuropathic upper back pain and hyperpigmented skin lesions. Neural therapy may be an alternative option that can be successfully applied in the treatment of these patients