Dergiler / Journal of Cellular Neuroscience and Oxidative Stress / 2021 / Cilt: 13 - Sayı: 1

Quantitative description of publications (1986-2020) related to Alzheimer disease and oxidative stress: A bibliometric study

Sayfa
971–984
Yayın Tarihi
2021-09-20
DOI
—
🏛️Bu makalenin bibliyografik verileri ve tam metin bağlantısı DergiPark üzerinden sağlanmıştır.
DergiPark Kaydı ↗
✨

Yapay Zeka Hızlı Bakış & Çıkarımlar

AI Insight
⚡ TL;DR(1 Cümlede Hızlı Çıkarım)

Our findings highlight current hot topics related to biomarkers for screening and diagnosis of AD as well as neuroprotective agents used as disease-modifying therapies of AD.

📌 3 Maddede Bu Makale

🎯
Amaç & Hipotez:While the pathological mechanism of Alzheimer’s Disease (AD) is unclear, oxidative stress has been proposed to be one of its related theories, which can help to uncover the disease’s pathological factors.
📊
Temel Bulgular & Veri:Based on the keywords used, which are related to oxidative stress and AD in the article title, 996 documents were retrieved for further analysis.
💡
Sonuç & Katkı:Our findings highlight current hot topics related to biomarkers for screening and diagnosis of AD as well as neuroprotective agents used as disease-modifying therapies of AD.
🔬Tür:Literatür & Derleme Çalışması
📍Kapsam:Genişletilmiş Akademik Örneklem
⚙️Değişkenler:Alzheimer's disease · Oxidative stress · Bibliometric

Abstract

While the pathological mechanism of Alzheimer’s Disease (AD) is unclear, oxidative stress has been proposed to be one of its related theories, which can help to uncover the disease’s pathological factors. This review aims to provide a quantitative description and data visualisation of oxidative stress and AD research from the literature obtained from the Scopus database. Based on the keywords used, which are related to oxidative stress and AD in the article title, 996 documents were retrieved for further analysis. Microsoft Excel, VOSviewer, and Harzing’s Publish or Perish were used to conduct the frequency analysis, data visualization, and citation analysis. There is a continuous growth in the number of publications on research in oxidative stress and AD, starting from 1986 and spanning 35 years. The most cited article was “Oxidative stress hypothesis in Alzheimer's disease”. The Journal of Alzheimer's Disease published the most number of publications related to oxidative stress and AD, while the United States and its institutions were the main contributors. Our findings suggest that research on aetiopathology, biomarkers, and neuroprotective agents for AD dominated this research field. Our bibliometric analysis provides distinct trends in oxidative stress and AD research in the last 35 years. Our findings highlight current hot topics related to biomarkers for screening and diagnosis of AD as well as neuroprotective agents used as disease-modifying therapies of AD.

🔖

Bu Makaleye Atıf Yap

QAID, E. Y. A., LONG, I., AZMAN, K. F., AHMAD, A. H., OTHMAN, Z., SIRAJUDEEN, K., AHMİ, A., ZAKARIA, R. (2021). Quantitative description of publications (1986-2020) related to Alzheimer disease and oxidative stress: A bibliometric study. Journal of Cellular Neuroscience and Oxidative Stress, Cilt: 13 - Sayı: 1, 971–984. https://ajindex.com/articles/827242

📄 Tam Metin PDF Okuyucu

📄

PDF görüntüleyici hazırlanıyor...

Kaynakça (12)

  1. Ahmed T, Gilani AH. (2014). Therapeutic potential of turmeric in Alzheimer's disease: curcumin or curcuminoids? Phytother Res. 28(4):517-525.
  2. Akbar H, Duan X, Saleem S, Davis AK, Zheng Y. (2016). RhoA and Rac1 GTPases differentially regulate agonist-receptor mediated reactive oxygen species generation in platelets. PLOS One. 11:e0163227. Alzheimer's disease facts and figures. (2021). Alzheimers Dement. 17(3):327-406.
  3. Atack JR, Perry EK, Bonham JR, Candy JM, Perry RH. (1987). Molecular forms of butyrylcholinesterase in the human neocortex during development and degeneration of the cortical cholinergic system. J Neurochem. 48:1687-1692.
  4. Avramopoulos D. (2009). Genetics of Alzheimer's disease: recent advances. Genome Med. 1(3):34. Blennow K, Zetterberg H. (2018). Biomarkers for Alzheimer's disease: current status and prospects for the future. J Intern Med. 284(6):643-663.
  5. Burns A, Iliffe S. (2009). Alzheimer's disease. BMJ. 338:b158. Burns A, Jacoby R, Levy R. (1990). Psychiatric phenomena in Alzheimer's disease. IV: disorders of behaviour. Br J Psychiatry. 157:86-94.
  6. Butterfield DA. (2002). Amyloid beta-peptide (1-42)-induced oxidative stress and neurotoxicity: implications for neurodegeneration in Alzheimer's disease brain. A review. Free Radic Res. 36(12):1307-1313.
  7. Butterfield DA, Castegna A, Lauderback CM, Drake J. (2002). Evidence that amyloid beta-peptide-induced lipid peroxidation and its sequelae in Alzheimer's disease brain contribute to neuronal death. Neurobiol Aging. 23(5):655-664.
  8. Butterfield DA, Lauderback CM. (2002), Lipid peroxidation and protein oxidation in Alzheimer's disease brain: potential causes and consequences involving amyloid beta-peptide-associated free radical oxidative stress. Free Radic Biol Med. 32(11):1050-1060.
  9. Butterfield DA, Poon HF, Clair DS, Keller JN, Pierce WM, Klein JB, Markesbery WR. (2006). Redox proteomics identification of oxidatively modified hippocampal proteins in mild cognitive impairment: Insights into the development of Alzheimer’s disease. Neurobiol Dis. 22:223-232.
  10. Cheignon C, Tomas M, Bonnefont-Rousselot D, Faller P, Hureau C, Collin F. (2018). Oxidative stress and the amyloid beta peptide in Alzheimer's disease. Redox Biol. 14:450-464.
  11. Chen H, Wan Y, Jiang S, Cheng Y. (2014). Alzheimer’s disease research in the future: bibliometric analysis of cholinesterase inhibitors from 1993 to 2012. Scientometrics. 98:1865-1877.
  12. Christen Y. (2000). Oxidative stress and Alzheimer disease. Am J Clin Nutr. 71(2):621S-6