Dergiler / Eurasian Journal of Medical Investigation / 2020 / Cilt: 4 - Sayı: 2

Electrocardiographic Strain Sign as a Symptom Equivalent for the Severity of Aortic Stenosis

Sayfa
217–220
Yayın Tarihi
2022-10-12
DOI
—
🏛️Bu makalenin bibliyografik verileri ve tam metin bağlantısı DergiPark üzerinden sağlanmıştır.
DergiPark Kaydı ↗
✨

Yapay Zeka Hızlı Bakış & Çıkarımlar

AI Insight
⚡ TL;DR(1 Cümlede Hızlı Çıkarım)

The relationship betweenstrain and symptoms availability was found statistically important (p

📌 3 Maddede Bu Makale

🎯
Amaç & Hipotez:Objectives: Aortic stenosis is a disorder characterized by inflammation, atherosclerosis, and calcium deposition in theaortic valve.
📊
Temel Bulgular & Veri:The asymptomatic patients with severe aortic stenosis with normal left ventricular function (C1 stage)have been recommended clinical monitoring according to American guidelines.
💡
Sonuç & Katkı:The relationship betweenstrain and symptoms availability was found statistically important (p
🔬Tür:Araştırma Makalesi
📍Kapsam:2014 Verileri
⚙️Değişkenler:Alan Parametreleri

Abstract

Objectives: Aortic stenosis is a disorder characterized by inflammation, atherosclerosis, and calcium deposition in theaortic valve. The asymptomatic patients with severe aortic stenosis with normal left ventricular function (C1 stage)have been recommended clinical monitoring according to American guidelines. However, symptom definition by thepatients may be challenging. Besides, the symptom-dependent decision for the selection of the therapy can be latefor valve intervention. In this study, we evaluated the use of electrocardiographic strain sign as equivalent of aorticstenosis symptoms.Methods: In this retrospective study, 102 consecutive patients with AS were examined between October 2014 to September2019. According to electrocardiographic strain sign, the patients were divided into group I (strain sign exist)and group II (without strain sign). These groups were studied in terms of trans-aortic gradient and symptoms (angina,syncope, and dyspnea).Results: The relationship between strain availability and higher gradient was found statistically important. Strain signsensitivity and specificity in prediction ≥80 mmHg gradient had 76.3% and 83.3%, respectively. The relationship betweenstrain and symptoms availability was found statistically important (p

🔖

Bu Makaleye Atıf Yap

SARIÇAM, E., ÖCAL, A. (2022). Electrocardiographic Strain Sign as a Symptom Equivalent for the Severity of Aortic Stenosis. Eurasian Journal of Medical Investigation, Cilt: 4 - Sayı: 2, 217–220. https://ajindex.com/articles/508639

📄 Tam Metin PDF Okuyucu

📄

PDF görüntüleyici hazırlanıyor...

Kaynakça (14)

  1. Owens DS, Katz R, Takasu J, Kronmal R, Budoff MJ, O’Brien KD. Incidence and progression of aortic valve calcium in the Multi-Ethnic Study of Atherosclerosis (MESA). Am J Cardiol 2010
  2. 701–8. [CrossRef ] 2. Nishimura RA, Otto CM, Bonow RO, et al. 2014 AHA/ACC Guideline for the Management of Patients With Valvular Heart Disease: executive summary
  3. a report of the American College of Cardiology/American Heart Association Task Force on Practice Guidelines. Circulation 2014
  4. 2440–92. [CrossRef ] 3. Kanwar A, Thaden JJ, Nkomo VT. Management of Patients with Aortic Valve Stenosis. Mayo Clin Proc 2018
  5. 488–508. 4. Hein S, Arnon E, Kostin S, Schönburg M, Elsässer A, Polyakova V, Bauer EP, Klövekorn WP, Schaper J. Progression from compensated hypertrophy to failure in the pressure-overloaded human heart: structural deterioration and compensatory mechanisms. Circulation 2003
  6. 984–91. [CrossRef ] 5. Chin CW, Vassiliou V, Jenkins WS, Prasad SK, Newby DE, Dweck MR. Markers of left ventricular decompensation in aortic stenosis. Expert Rev Cardiovasc Ther 2014
  7. 1–12. [CrossRef ] 6. Rautaharju PM, Surawicz B, Gettes LS, Bailey JJ, Childers R, Deal BJ et al. AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: part IV: the ST segment, T and U waves, and the QT interval: a scientific statement from the American Heart Association Electrocardiography and Arrhythmias Committee, Council on Clinical Cardiology
  8. the American College of Cardiology Foundation
  9. and the Heart Rhythm Society. Endorsed by the International Society for Computerized Electrocardiology. J Am Coll Cardiol 2009
  10. 982–91. [CrossRef ] 7. Lombard JT, Selzer A. Valvular aortic stenosis. A clinical and hemodynamic profile of patients. Ann Intern Med 1987
  11. 292– 8. [CrossRef ] 8. Bertrand ME, LaBlanche JM, Tilmant PY, et al. Coronary sinus blood flow at rest and during isometric exercise in patients with aortic valve disease. Mechanism of angina pectoris in presence of normal coronary arteries. Am J Cardiol 1981
  12. [CrossRef ] 9. Marcus ML. Donald BD, Horatzka LF, Wright CB, Eastham CL. Decreased coronary reserve. A mechanism for angina pectoris in patients with aortic stenosis and normal coronary arteries. N Engl J Med 1982
  13. 1362–6. [CrossRef ] 10. Gould KL. Why angina pectoris in aortic stenosis? Circulation 1997
  14. [CrossRef ] 11. Richards AM, Ikram H, Nicholls MG, Hamilton EJ, Richards RD. Syncope in aortic valvular stenosis. Lancet 1