Dergiler / Turkish Journal of Hematology / 2018 / Cilt: 35 - Sayı: 1

Cyclic Guanosine Monophosphate-Dependent Protein Kinase I Stimulators and Activators Are Therapeutic Alternatives for Sickle Cell Disease

Sayfa
89–90
DOI
—
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Bu Makaleye Atıf Yap

GHANTA, M., PANCHANATHAN, E., LAKKAKULA, B. V. (2018). Cyclic Guanosine Monophosphate-Dependent Protein Kinase I Stimulators and Activators Are Therapeutic Alternatives for Sickle Cell Disease. Turkish Journal of Hematology, Cilt: 35 - Sayı: 1, 89–90. https://ajindex.com/articles/413399

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Kaynakça (10)

  1. Verma H, Mishra H, Khodiar PK, Patra PK, Bhaskar LV. NOS3 27-bp and IL4 70-bp VNTR polymorphisms do not contribute to the risk of sickle cell crisis. Turk J Hematol 2016;33:365-366
  2. Schlossmann J, Ammendola A, Ashman K, Zong X, Huber A, Neubauer G, Wang GX, Allescher HD, Korth M, Wilm M, Hofmann F, Ruth P. Regulation of intracellular calcium by a signalling complex of IRAG, IP3 receptor and cGMP kinase Iβ. Nature 2000;404:197-201
  3. Weiner DL, Hibberd PL, Betit P, Cooper AB, Botelho CA, Brugnara C. Preliminary assessment of inhaled nitric oxide for acute vaso-occlusive crisis in pediatric patients with sickle cell disease. JAMA 2003;289:1136- 1142
  4. Sharma D, Potoka K, Kato GJ. Nitric oxide, phosphodiesterase inhibitors and soluble guanylate cyclase stimulators as candidate treatments for sickle cell disease. Journal of Sickle Cell Disease and Hemoglobinopathies 2016:JSCDH-D-16-00097
  5. Mancuso C, Scapagini G, Curro D, Giuffrida Stella AM, De Marco C, Butterfield DA, Calabrese V. Mitochondrial dysfunction, free radical generation and cellular stress response in neurodegenerative disorders. Front Biosci 2007;12:1107-1123
  6. Ghanta M, Panchanathan E, Lakkakula BVKS, Narayanaswamy A. Retrospection on the role of soluble guanylate cyclase in Parkinson’s disease. J Pharmacol Pharmacother 2017;8:87-91
  7. Li Y, Maher P, Schubert D. Requirement for cGMP in nerve cell death caused by glutathione depletion. J Cell Biol 1997;139:1317-1324
  8. Antl M, von Brühl ML, Eiglsperger C, Werner M, Konrad I, Kocher T, Wilm M, Hofmann F, Massberg S, Schlossmann J. IRAG mediates NO/cGMPdependent inhibition of platelet aggregation and thrombus formation. Blood 2007;109:552-559
  9. Ikuta T, Ausenda S, Cappellini MD. Mechanism for fetal globin gene expression: role of the soluble guanylate cyclase-cGMP-dependent protein kinase pathway. Proc Natl Acad Sci USA 2001;98:1847-1852
  10. Moon TM, Tykocki NR, Sheehe JL, Osborne BW, Tegge W, Brayden JE, Dostmann WR. Synthetic peptides as cGMP-independent activators of cGMP-dependent protein kinase Iα. Chem Biol 2015;22:1653-1661