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Dergiler / Turkish Journal of Veterinary and Animal Sciences / 2008 / Cilt: 32 - Sayı: 4

Anticoagulant Rodenticide Intoxication in Animals – A Review

İvan VALCHEV
Rumen BINEV
Veska YORDANOVA
Yordan NIKOLOV
Sayfa
237–243
Yayın Tarihi
2008-04-01
DOI
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⚡ TL;DR(1 Cümlede Hızlı Çıkarım)

The general prophylaxis consists of placing baits out of reach of animals, daily control of baits and dead rodents, which are to be timely removed.

📌 3 Maddede Bu Makale

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Amaç & Hipotez:The newest measures for the control of harmful rodent populations are from the anticoagulant rodenticide group, which are divided into 2 subgroups: first and second generations, and indandione derivatives.
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Temel Bulgular & Veri:Anticoagulant rodenticides inhibit the enzyme vitamin K-dependent carboxylase and thus impair the reactivation of vitamin K1, indirectly affecting physiological blood coagulation.
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Sonuç & Katkı:The general prophylaxis consists of placing baits out of reach of animals, daily control of baits and dead rodents, which are to be timely removed.
🔬Tür:Araştırma Makalesi
📍Kapsam:Genişletilmiş Akademik Örneklem
⚙️Değişkenler:Intoxication · anticoagulant rodenticides · animals

Özet

The newest measures for the control of harmful rodent populations are from the anticoagulant rodenticide group, which are divided into 2 subgroups: first and second generations, and indandione derivatives. Non-target organisms are potentially at risk of direct consumption of baits (primary hazard) and of eating poisoned rodents (secondary hazard). Anticoagulant rodenticides inhibit the enzyme vitamin K-dependent carboxylase and thus impair the reactivation of vitamin K1, indirectly affecting physiological blood coagulation. The diagnosis is made on the basis of clinical signs (massive hemorrhages), laboratory findings, and especially the changes in coagulation markers (APTT, PT, TT, PCT, ACT, FDPs, and PIVKA). The specific antidote is vitamin K1. The general prophylaxis consists of placing baits out of reach of animals, daily control of baits and dead rodents, which are to be timely removed.

Abstract

The newest measures for the control of harmful rodent populations are from the anticoagulant rodenticide group, which are divided into 2 subgroups: first and second generations, and indandione derivatives. Non-target organisms are potentially at risk of direct consumption of baits (primary hazard) and of eating poisoned rodents (secondary hazard). Anticoagulant rodenticides inhibit the enzyme vitamin K-dependent carboxylase and thus impair the reactivation of vitamin K1, indirectly affecting physiological blood coagulation. The diagnosis is made on the basis of clinical signs (massive hemorrhages), laboratory findings, and especially the changes in coagulation markers (APTT, PT, TT, PCT, ACT, FDPs, and PIVKA). The specific antidote is vitamin K1. The general prophylaxis consists of placing baits out of reach of animals, daily control of baits and dead rodents, which are to be timely removed.

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Bu Makaleye Atıf Yap

VALCHEV, İ., BINEV, R., YORDANOVA, V., NIKOLOV, Y. (2008). Anticoagulant Rodenticide Intoxication in Animals – A Review. Turkish Journal of Veterinary and Animal Sciences, Cilt: 32 - Sayı: 4, 237–243. https://ajindex.com/articles/382931

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Kaynakça (8)

  1. Litovitz, T.L., Klein-Schwartz, W., Dyer, K.S., Shannon, M., Lee, S., Powers, M.:: 1997 annual report of the American Association of Poison Control Centers Toxic Exposure Surveillance System. Am. J. Emerg. Med., 1998; 16: 443-497.
  2. Maroni, M., Colosio, C., Ferioli, A., Fait, A.: Biological monitoring of pesticide exposure: a review. Toxicology, 2000; 143: 1-118. 3. Morrissey, B., Burgess, J.L, Robertson, W.O.: Washington's experience and recommendations Re: anticoagulant rodenticides. Vet. Hum. Toxicol., 1995; 37: 362-363. 4. Murphy, M.J.: Rodenticides. Vet. Clin. North Am. Small Anim. Pract., 2002; 32: 469-484. 5. Dorman, D.C., Simon, J., Harlin, K.A., Buck, W.B.: Diagnosis of bromethalin toxicosis in the dog. J. Vet. Diagn. Invest., 1990; 2: 123-128. 6. Dorman, D.C., Zachary, J.F., Buck, W.B.: Neuropathologic Endepols, S., Klemann, N., Pelz, H.J., Ziebell, K.L.: A scheme for the placement of rodenticide baits for rat eradication on confinement livestock farms. Prev. Vet. Med., 2003; 58: 115- 123. Fournier-Chambrillon, C., Berny, P.J., Coiffier, O., Barbedienne, P., Dassé, B., Delas, G., Galineau, H., Mazet, A., Pouzenc, P., Rosoux, R., Fournier, P.: Evidence of secondary poisoning of free-ranging riparian mustelids by anticoagulant rodenticides in France: implications for conservation of European mink (Mustela lutreola). J. Wildl. Dis., 2004; 40: 688-695.
  3. Berny, P.J., Buronfosse, T., Buronfosse, F., Lamarque, F., Lorgue, G.: Field evidence of secondary poisoning of foxes (Vulpes vulpes) and buzzards (Buteo buteo) by bromadiolone, a 4-year survey. Chemosphere, 1997; 35: 1817-1829.
  4. Binev, R., Petkov, P., Rusenov, A.: Intoxication with anticoagulant rodenticide bromadiolone in a dog – a case report. Vet. Arhiv, 2005; 75: 273-282.
  5. Blocker, T.L., Roberts, B.K.: Acute tracheal obstruction associated with anticoagulant rodenticide intoxication in a dog. J. Small Anim. Pract., 1999; 40: 577-580.
  6. Boermans, H.J., Johnstone, I., Black, W.D., Murphy, M.: Clinical signs, laboratory changes and toxicokinetics of brodifacoum in the horse. Can. J. Vet. Res., 1991; 55: 21-27.
  7. Haug, B., Schjİdt-Iversen, L., Rygh, J.: Poisoning with long-acting anticoagulants Tidsskr. Nor. Laegeforen., 1992; 112: 1958- 1960. (article in Norwegian with an abstract in English) Hansen, N., Beck, C.: Bilateral hydronephrosis secondary to anticoagulant rodenticide intoxication in a dog. J. Vet. Emerg. Crit. Care, 2003; 13: 103-107.
  8. James, S.B., Raphael, B.L., Cook, R.A.: Brodifacoum toxicity and