Journals / Journal of Surgery and Medicine / 2021 / Cilt: 5 - Sayı: 5

A rare cause of hyperamylasemia: A case with pneumothorax and subcutaneous emphysema around the parotid gland in a trauma patient

Pages
553–555
Publication Date
2021-05-01
DOI
—
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AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

Possible mechanisms of hyperamylasemia in our case are acute hypoxia-induced hyperamylasemia because of left lung pneumothorax and massage-like activity of subcutaneous emphysema around the parotid gland.

📌 3 Key Takeaways

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Objective & Hypothesis:Amylase is a common laboratory test in emergency departments, especially in trauma patients.
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Key Findings & Data:Although hyperamylasemia is mostly caused by injury to the pancreas in trauma patients, non-pancreatic causes of hyperamylasemia are not rare.
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Conclusion & Impact:Possible mechanisms of hyperamylasemia in our case are acute hypoxia-induced hyperamylasemia because of left lung pneumothorax and massage-like activity of subcutaneous emphysema around the parotid gland.
🔬Design:Research Article
📍Scope:Comprehensive Sample
⚙️Variables:Hyperamylasemia · Acute pancreatitis · Pneumothorax

Abstract

Amylase is a common laboratory test in emergency departments, especially in trauma patients. Although hyperamylasemia is mostly caused by injury to the pancreas in trauma patients, non-pancreatic causes of hyperamylasemia are not rare. Being aware of these causes may prevent unnecessary diagnostic tests. In this study, we presented a trauma patient with hyperamylasemia of non-pancreatic origin. Possible mechanisms of hyperamylasemia in our case are acute hypoxia-induced hyperamylasemia because of left lung pneumothorax and massage-like activity of subcutaneous emphysema around the parotid gland.

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Cite This Article

PEKSÖZ, R., ASYA, O. (2021). A rare cause of hyperamylasemia: A case with pneumothorax and subcutaneous emphysema around the parotid gland in a trauma patient. Journal of Surgery and Medicine, Cilt: 5 - Sayı: 5, 553–555. https://ajindex.com/articles/779958

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References (7)

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