Journals / İstanbul Kuzey Klinikleri / 2019 / Cilt: 6 - Sayı: 1

Diagnosis and treatment of urticaria in primary care

Pages
93–99
Publication Date
2022-11-04
DOI
—
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AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

H1 antihistamines and, sometimes, short-term systemic corticosteroids arepreferred for the treatment; H2 antagonists may be added during resistant cases, although other treatment options, such asomalizumab, cyclosporine, and leukotriene receptor antagonists, may be considered during missed events.

📌 3 Key Takeaways

🎯
Objective & Hypothesis:Urticaria, also known as hives among people, is a very common disease characterized by erythematous, edematous, itchy,and transient plaques that involve skin and mucous membranes.
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Key Findings & Data:The lesions usually resolve spontaneously within 2–3 h without a trace.
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Conclusion & Impact:H1 antihistamines and, sometimes, short-term systemic corticosteroids arepreferred for the treatment; H2 antagonists may be added during resistant cases, although other treatment options, such asomalizumab, cyclosporine, and leukotriene receptor antagonists, may be considered during missed events.
🔬Design:Research Article
📍Scope:Comprehensive Sample
⚙️Variables:Domain Parameters

Abstract

Urticaria, also known as hives among people, is a very common disease characterized by erythematous, edematous, itchy,and transient plaques that involve skin and mucous membranes. It is classified as acute spontaneous urticaria, chronic spontaneous urticaria, chronic inducible urticaria, and episodic chronic urticaria. Many factors such as infections, medicines, food,psychogenic factors, and respiratory allergens are accused of etiology, but sometimes, it is idiopathic. Clinical presentationinvolves red, swelling, and itchy plaques. The lesions usually resolve spontaneously within 2–3 h without a trace. The patientsare sometimes confronted with an angioedema that can also involve the respiratory tract. In this case mucous membranes,such as eyelids, lips, swell with some pain and burning sensation. If respiratory tracts are involved, it may be life threateningand should be treated urgently. The diagnosis is usually straightforward, urticarial vasculitis, drug eruptions, viral eruptions,and urticaria pigmentosa must also be considered. H1 antihistamines and, sometimes, short-term systemic corticosteroids arepreferred for the treatment; H2 antagonists may be added during resistant cases, although other treatment options, such asomalizumab, cyclosporine, and leukotriene receptor antagonists, may be considered during missed events.

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Cite This Article

AKDENİZ, N., KAYIRAN, M. A. (2022). Diagnosis and treatment of urticaria in primary care. İstanbul Kuzey Klinikleri, Cilt: 6 - Sayı: 1, 93–99. https://ajindex.com/articles/652018

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References (20)

  1. Darlenski R, Kazandjieva J, Zuberbier T, Tsankov N. Chronic urticaria as a systemic disease. Clin Dermatol 2014
  2. 420–3. [CrossRef ] 2. Sánchez-Borges M, Asero R, Ansotegui IJ, Baiardini I, Bernstein JA, Canonica GW, et al
  3. WAO Scientific and Clinical Issues Council. Diagnosis and treatment of urticaria and angioedema: a worldwide perspective. World Allergy Organ J 2012
  4. 125–47. [CrossRef ] 3. Zuberbier T, Aberer W, Asero R, Bindslev-Jensen C, Brzoza Z, Canonica GW, et al
  5. European Academy of Allergy and Clinical Immunology
  6. Global Allergy and Asthma European Network
  7. European Dermatology Forum
  8. World Allergy Organization. The EAACI/GA(2) LEN/ EDF/WAO Guideline for the definition, classification, diagnosis, and management of urticaria: the 2013 revision and update. Allergy 2014
  9. 868–87. [CrossRef ] 4. Önder M. Ürtiker ve anjioödem, genel yaklaşım ve sınıflandırma. Turkiye Klinikleri J Dermatol-Special Topics 2008
  10. 6–11. 5. Wedi B. Urticaria. J Dtsch Dermatol Ges 2008
  11. 306–17. [CrossRef ] 6. Sabroe RA, Greaves MW. The pathogenesis of chronic idiopathic urticaria. Arch Dermatol 1997
  12. 1003–8. [CrossRef ] 7. Aslan Kayıran M, Akdeniz N. Intracutaneous tests (Type 1 allergy test, Type 4 allergy test). Turkiye Klinikleri J Dermatol-Special Topics 2017
  13. 13–8. 8. Kocatürk Göncü E, Aktan Ş, Atakan N, Bülbül Başkan E, Erdem T, Koca R et al. Türkiye Ürtiker Tanı ve Tedavi Kılavuzu-2016. Turkderm- Arch Turk Dermatol Venerology 2016
  14. 82–98. 9. Sampson HA. Adverse reactions to foods. In: Adkinson NF Jr., Yunginger JW, Busse WW, Bochner BS, Holgate ST, Simons ER, editors. Middleton’s Allergy Principles and Practice. 6th ed. Philadelphia: Mosby
  15. p. 1619–43. 10. Rajan JP, Simon RA, Bosso JV. Prevalence of sensitivity to food and drug additives in patients with chronic idiopathic urticaria. J Allergy Clin Immunol Pract 2014
  16. 168–71. [CrossRef ] 11. Chow SK. Management of chronic urticaria in Asia: 2010 AADV consensus guidelines. Asia Pac Allergy 2012
  17. 149–60. [CrossRef ] 12. Bülbül Başkan E. Etiology and Pathogenesis of Chronic Urticaria. Turkiye Klinikleri J Dermatol-Special Topics 2015
  18. 13–9. 13. Aktan Ş. Physical Urticarias: Pathogenesis and Diagnostic Testing Turkiye Klinikleri J Dermatol-Special Topics 2015
  19. 34–40. 14. Sánchez-Borges M, Capriles-Hulett A, Caballero-Fonseca F, GonzálezAveledo L. Urticaria in children attending allergy services. [Article in Spanish]. Rev Alerg Mex 2014
  20. 90–8. 15. Staubach P, Dechene M, Metz M, Magerl M, Siebenh