ACUTE EFFECT OF CIGARETTE SMOKING ON VENTRICULAR REPOLARIZATION PARAMATERS

Objective & Hypothesis:Background: Acute cigarette smoking has been reported to increase heart rate and blood pressure as a consequence of sympathetic stimulation.

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Key Findings & Data:A 12-lead surface ECG recorded at a paper speed of 50 ms/s was obtained from all participants before and average 15 min. after cigarette smoking.

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Conclusion & Impact:Hart rate and RRV2 significantly increased after smoking a cigarette (67±7 vs. 72±8 beat/min, 984±169ms vs. 875±150ms, respectively p

🔬Design:Research Article

📍Scope:Comprehensive Sample

⚙️Variables:Cigarette smoking · ventricular repolarization · and arrhythmia

Abstract

Background: Acute cigarette smoking has been reported to increase heart rate and blood pressure as a consequence of sympathetic stimulation. In this study we sought to determine if single dose cigarette smoking influences ventricular repolarization paramaters in habitual smokers. Method and Results: The study population consisted of twenty-four long-term heavy smokers. A 12-lead surface ECG recorded at a paper speed of 50 ms/s was obtained from all participants before and average 15 min. after cigarette smoking. Maximum QT (QTmax), minimum QT (QTmin) intervals, QT dispersion (QTd) and corrected QT dispersion (QTcd) were calculated on each ECG record. QT and RR intervals in V2 leads (QTV2 and RRV2) were also measured before and after smoking. Hart rate and RRV2 significantly increased after smoking a cigarette (67±7 vs. 72±8 beat/min, 984±169ms vs. 875±150ms, respectively p

Özet

Background: Acute cigarette smoking has been reported to increaseheart rate and blood pressure as a consequence of sympatheticstimulation. In this study we sought to determine if single dosecigarette smoking influences ventricular repolarization paramatersin habitual smokers.Method and Results:The study population consisted of twenty-fourlong-term heavy smokers. A 12-lead surface ECG recorded at apaper speed of 50 ms/s was obtained from all participants before andaverage 15 min. after cigarette smoking. Maximum QT (QTmax),minimum QT (QTmin) intervals, QT dispersion (QTd) andcorrected QT dispersion (QTcd) were calculated on each ECGrecord. QT and RR intervals in V2 leads (QTV2 and RRV2) werealso measured before and after smoking. Hart rate and RRV2significantly increased after smoking a cigarette (67±7 vs. 72±8beat/min, 984±169ms vs. 875±150ms, respectively p<0.05).However, the other parameters including QTmax, QTmin, QTd,QTcd and QTV2 did not alter compared to baseline values.Concusion:Single dose cigarette smoking does not seem to beassociated with ventricular heterogeneity both in habitual smokersalthough it leads to sympathetic hyperactivity.

Keywords

Cigarette smoking ventricular repolarization and arrhythmia andarrhythmia.

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Cite This Article

Karakaya, O., SAGLAM, M., Esen, A. M., BARUTCU, I., Barutcu, I., OZDEMIR, N., YAYMACI, B., Esen, O., BOZTOSUN, B., Boztosun, B., Kaymaz, C. (2005). ACUTE EFFECT OF CIGARETTE SMOKING ON VENTRICULAR REPOLARIZATION PARAMATERS. Koşuyolu Heart Journal, Cilt: 9 - Sayı: 1, 1–7. https://ajindex.com/articles/64168

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