SavedAdd your journal

Journals / Türkiye Fiziksel Tıp ve Rehabilitasyon Dergisi / 2004 / Cilt: 50 - Sayı: 1

Effects of atherosclerotic risk factors on the clinical findings and prognosis of lumbar spinal stenosis

Aterosklerotik risk faktörlerinin lomber spinal stenozun klinik bulgularına ve prognoza etkisi

Burak TANGÜREK
Aydın Saliha TANGÜREK
Günay CAN
Hidayet SARI
Pages
3–9
Publication Date
2022-11-05
DOI
—
🏛️Bibliographic metadata and full text access for this article are provided via DergiPark.
DergiPark Record ↗
✨

AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

Walking distances were shorter and neurogenic complaints were higher in patients with ASVD before treatment (p

📌 3 Key Takeaways

🎯
Objective & Hypothesis:We investigated the clinical relationships between lumbar spinal stenosis (LSS) and atherosclerotic risk factors (ASRF) such as diabetes mellitus (DM), hypercholesterolemia (HC), hypertension (HT) and atherosclerotic vascular disease (ASVD) as well as the effects of these diseases to the results of conservative treatment.
📊
Key Findings & Data:Physical therapy were applied to all 38 patients.
💡
Conclusion & Impact:Walking distances were shorter and neurogenic complaints were higher in patients with ASVD before treatment (p
🔬Design:Research Article
📍Scope:Comprehensive Sample
⚙️Variables:Domain Parameters

Abstract

We investigated the clinical relationships between lumbar spinal stenosis (LSS) and atherosclerotic risk factors (ASRF) such as diabetes mellitus (DM), hypercholesterolemia (HC), hypertension (HT) and atherosclerotic vascular disease (ASVD) as well as the effects of these diseases to the results of conservative treatment. Presence of ASRF and ASVD, complaints of pain and neurogenic claudication (NC) were identified and physical inspection including walking distances, lumbar area inspection, vascular inspection were performed. Radiological and laboratory tests were evaluated. Physical therapy were applied to all 38 patients. Walking distances were shorter and neurogenic complaints were higher in patients with ASVD before treatment (p

Özet

Çalışmamızda lomber spinal stenoz (LSS) ile diabetes mellitus (DM), hipertansiyon (HT), hiperkolesterolemi (HK) gibi aterosklerotik risk faktörleri ve aterosklerotik damar hastalığı (ASDH) arasındaki klinik ilişkileri ve bu durumların konservatif tedavi sonuçlarına etkisini araştırdık. 38 LSS’lu hastanın anamnezleri (DM, HT, HK, ASDH, ilaç kullanımları, ağrı sorgulaması, nörojenik klodikasyon şikayetlerinin sorgulaması) alındı; başlangıçta ve tedavi sonrasında fizik muayeneleri (yürüme mesafeleri, lomber bölge muayenesi, damar muayenesi) ; laboratuvar ve radyolojik tetkikleri yapıldı. Hastalara 15 seans fizik tedavi uygulandı. Tedavi öncesinde; ASDH olanlarda, olmayanlara göre yürüme mesafeleri daha kısa ve nörojenik şikayetleri daha fazla bulundu (p 0,05). Sonuç olarak; LSS’da, HT ve ASDH varlığı tedaviye yanıtı olumsuz etkilemektedir. HK ve DM da ASDH riski taşıdıklarından, dolaylı olarak risk oluştururlar. Bu verilerle uyumlu olarak, ASDH ve atreosklerotik risk faktörleri varlığı LSS’lu hastalarda başlangıç şikayetlerine ve konservatif tedavi sonuçlarına olumsuz etki yapmaktadır. LSS tedavisine bu hastalıkların tedavisinin de eklenmesi uygundur

🔖

Cite This Article

TANGÜREK, B., TANGÜREK, A. S., CAN, G., SARI, H. (2022). Effects of atherosclerotic risk factors on the clinical findings and prognosis of lumbar spinal stenosis. Türkiye Fiziksel Tıp ve Rehabilitasyon Dergisi, Cilt: 50 - Sayı: 1, 3–9. https://ajindex.com/articles/629286

📄 Full Text PDF Viewer

📄

PDF görüntüleyici hazırlanıyor...

References (18)

  1. Arnoldi CC, Brodsky AE, Cachoix J, Crock HV, et al. Lumbar spinal stenosis and nerve root entrapment. Syndromes,definition and classification. Clin Orthop 1976; 115: 4-5
  2. Kirkaldy WK, Melvor GWD. Editorial Comment: Lumbar spinal stenosis. Clin Orthop 1976; 115: 1-2
  3. Hall S, Lowthian PJ. Lumbar spinal stenosis. In Klippel JH, Dieppe PA ed(s). Rheumatology. London: Mosby, 2000: 4.5.1
  4. Garfin SR, Rydevik BL, Lipson SJ. Spinal stenosis. In Rothman RH, Simeone FA ed(s).The Spine 3th edition. Philadelphia: Saunders Company, 1992: 791-824
  5. Tüzün Ş. Bel ve bacak ağrıları. Tüzün F, Eryavuz M, Akarırmak Ü ed(s). Hareket sistemi hastalıkları. İstanbul: Nobel, 1997: 245-260
  6. Onel D, Sarı H, Dönmez Ç. Lumbar spinal stenosis:clinical/radiologic therapeutic evaluation in 145 patients. Spine 1993; 18: 291-298
  7. Katz JN, Dalgas M, Stucki G, Lipson SG. Degenerative lumbar spinal stenosis. Diagnostic value of the history and physical examination. Arthritis Rheum 1995; 38: 1236-1241
  8. Andersson GBJ, McNeill TW. Definition and classification of lumbar spinal stenosis. In Andersson GBJ, McNeill TW ed(s). Lumbar spinal stenosis. St Louis: Mosby-Year Book, 1992: 9-15
  9. Hansson GK, Nilsson J. Pathogenesisi of atherosclerosis. In Crawford MH, Dimarco JP ed(s). Cardiology. London: Mosby, 2001: 1.1-1.12
  10. Libby P.Vascular disease. In Fauci AS, Braunwald E, Isselbacher KJ, Wilson JD, Martin JB, Kasper DL, Hause SL, Longo DL ed(s).Harrison’s principles of internal medicine.United States:Mc Graw-Hill, 1998:1345-1352
  11. Soydan İ. Etyolojik faktörler. İnflamasyon ve ateroskleroz InKültürsay H ed. Koroner kalp hastalığı:Primer ve sekonder korunma. İstanbul: ARGOS, 2001: 67-81
  12. Kaplan NM. Klinik hipertansiyon. Çeviri. İstanbul: Turgut yayıncılık;1998: 46
  13. Korkmaz M.Kardiovasküler risk faktörleri. In Arık N, Korkmaz M ed(s). Hipertansiyon. İstanbul: Format Matbaacılık, 1999: 46-73
  14. Soydan İ. Etyolojik faktörler. Risk faktörleri. In Kültürsay H ed(s). Koroner kalp hastalığı: Primer ve sekonder korunma. İstanbul: ARGOS, 2001: 135-138
  15. Lee JH, McCarty R. Pain threshold in diabetic rats:effects of good versus poor diabetic control.Pain 1992; 50: 231-236
  16. Blau SN, Logue V.Intermittent claudication of the cauda equina. Lancet 1961; 2: 1081-1086
  17. Evans JG. Neurogenic intermittent claudication. Br Med J 1964; 2: 985-987
  18. Raff MC, Sangalang V, Asbury AK. Ischemic mononeuropathy multiplex associated with diabetes mellitus. Arch.Neurol.1968; 18