The development of PRES in preeclampsia is discussed and the importance of prompt postpartum blood pressure control is emphasized. (J Turkish-German Gynecol Assoc 2010; 11: 216-9)
📌 3 Key Takeaways
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Objective & Hypothesis:We describe a 23 year old primigravid patient with severe preeclampsia complicated by posterior reversible encephalopathy syndrome (PRES), who presented with sensory and motor deficits and amnesia in the postpartum period Cranial magnetic resonance imaging (MRI) showed abnormal areas in the white matter of bilateral parieto-occipital lobes, indicating brain edema which disappeared completely on the follow-up scan taken four weeks after delivery together with complete symptom regression.
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Key Findings & Data:We describe a 23 year old primigravid patient with severe preeclampsia complicated by posterior reversible encephalopathy syndrome (PRES), who presented with sensory and motor deficits and amnesia in the postpartum period Cranial magnetic resonance imaging (MRI) showed abnormal areas in the white matter of bilateral parieto-occipital lobes, indicating brain edema which disappeared completely on the follow-up scan taken four weeks after delivery together with complete symptom regression.
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Conclusion & Impact:The development of PRES in preeclampsia is discussed and the importance of prompt postpartum blood pressure control is emphasized. (J Turkish-German Gynecol Assoc 2010; 11: 216-9)
🔬Design:Research Article
📍Scope:2010 Verileri
⚙️Variables:Domain Parameters
Abstract
We describe a 23 year old primigravid patient with severe preeclampsia complicated by posterior reversible encephalopathy syndrome (PRES), who presented with sensory and motor deficits and amnesia in the postpartum period Cranial magnetic resonance imaging (MRI) showed abnormal areas in the white matter of bilateral parieto-occipital lobes, indicating brain edema which disappeared completely on the follow-up scan taken four weeks after delivery together with complete symptom regression. The development of PRES in preeclampsia is discussed and the importance of prompt postpartum blood pressure control is emphasized. (J Turkish-German Gynecol Assoc 2010; 11: 216-9)
Özet
Bu yazıda 23 yaşında, ilk gebeliği olan şiddetli preeklamptik bir hastada postpartum dönemde duyusal ve motor sinir bulguları ve unutkanlık ile ortaya çıkan bir posterior reverzibl ensefalopati sendromu (PRES) sunulmuştur. Kranyal manyetik rezonans görüntüleme (MRI) ile bilateral parieto-occipital loblarda beyin ödemine işaret eden anormal alanlar görülmüş olup, doğumdan dört hafta sonra kontrol amaçlı yapılan görüntüleme testi ile bu lezyonların tamamen kaybolduğu ve semptomların da gerilediği tespit edilmiştir. Preeklampside PRES gelişimi tartışılarak, bu hastalarda postpartum kan basıncı kontrolünün önemi vurgulanmıştır. (J Turkish-German Gynecol Assoc 2010; 11: 216-9)
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Cite This Article
KAVAK, B., BAYKUŞ, Y., GÜRATEŞ, B., ÇELİK, H., AYGÜN, K. B., BERİLGEN, S. (2022). Ağır preeklampside posterior reverzibl ensefalopati sendromu: Olgu sunumu ve literatürün gözden geçirilmesi. Journal of the Turkish-German Gynecological Association, Cilt: 11 - Sayı: 4, 216–219. https://ajindex.com/articles/567776
Hinchey J, Chaves C, Appignani B, Breen J, Pao L, Wang A, et al. A reversible posterior leukoencephalopathy syndrome. N Engl J Med 1996; 334: 494-500
Striano P, Striano S, Tortora F, De Robertis E, Palumbo D, Elefante A, et al. Clinical spectrum and critical care management of Posterior Reversible Encephalopathy Syndrome (PRES). Med Sci Monit 2005; 11: 549-53
Negro A, Zuccoli G, Regolisti G, Mastrangeli S, Rossi E. Reversible posterior leukoencephalopathy associated with postpartum HELLP syndrome. Eur J Intern Med 2005; 16: 291-3
Peng WX, Nakaii M, Matsushima T, Asakura H. Atypical case of reversible posterior leucoencephalopathy syndrome associated with puerperal HELLP syndrome. Arch Gynecol Obstet 2008; 278: 269-71
Kastrup O, Maschke M, Wanke I, Diener HC. Posterior reversible encephalopathy syndrome due to severe hypercalcemia. J Neurol 2002; 249: 1563-6
Ay H, Buonanno FS, Schaefer PW, Le DA, Wang B, Gonzalez RG, et al. Posterior leukoencephalopathy without severe hypertension: utility of diffusion-weighted MRI. Neurology 1998; 51: 1369-76
Schwartz RB, Feske SK, Polak JF, DeGirolami U, Iaia A, Beckner KM, et al. Preeclampsia-eclampsia: clinical and neuroradiographic correlates and insights into the pathogenesis of hypertensive encephalopathy. Radiology 2000; 217: 371-6
Johansson BB. The blood-brain barrier and cerebral blood flow in acute hypertension. Acta Med Scand Suppl 1983; 678: 107-12
Edvinsson L, Owman C, Siesjö B. Physiological role of cerebrovascular sympathetic nerves in the autoregulation of cerebral blood flow. Brain Res 1976; 117: 519-23
Sheth RD, Riggs JE, Bodenstenier JB, Gutierrez AR, Ketonen LM, Ortiz OA. Parietal occipital edema in hypertensive encephalopathy: a pathogenic mechanism. Eur Neurol 1996; 36: 25-8
Sanders TG, Clayman DA, Sanchez-Ramos L, Vines FS, Russo L. Brain in eclampsia: MR imaging with clinical correlation. Radiology 1991; 180: 475-8
Bartynski WS, Boardman JF. Distinct imaging patterns and lesion distribution in posterior reversible encephalopathy syndrome. AJNR Am J Neuroradiol 2007; 28: 1320-7
Servillo G, Striano P, Striano S, Tortora F, Boccella P, De Robertis E, et al. Posterior reversi