Journals / Fiziksel Tıp ve Rehabilitasyon Bilimleri Dergisi / 2019 / Cilt: 22 - Sayı: 1

Treatment Resistance in Hypoxic Brain Injury Caused by Cardiopulmonary Arrest Due to an Allergic Anaphylaxis: Spasticity, Myoclonus or Both?

Alerjik Anafilaksiye Bağlı Kardiyopulmoner Arrest Sonucu Oluşan Hipoksik Beyin Hasarında Tedavi Direnci: Spasi site mi, Miyoklonus mu Yoksa Her İkisi de mi?

Pages
29–32
Publication Date
2022-10-18
DOI
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AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

Here, we present a case withHBI that complicated by spasticity and LAS because of both it is a rare condition and effected to rehabilitationprocess.

📌 3 Key Takeaways

🎯
Objective & Hypothesis:Anaphylaxis is an acute and life threatening reaction and often results death.
📊
Key Findings & Data:Withmedical developments, patients are more likely to survive after anaphylactic cardiopulmonary arrest.This leads to an increase in patients with hypoxic brain injury (HBI) who are in serious needof rehabilitation.
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Conclusion & Impact:Here, we present a case withHBI that complicated by spasticity and LAS because of both it is a rare condition and effected to rehabilitationprocess.
🔬Design:Research Article
📍Scope:Comprehensive Sample
⚙️Variables:Domain Parameters

Abstract

Anaphylaxis is an acute and life threatening reaction and often results death. Withmedical developments, patients are more likely to survive after anaphylactic cardiopulmonary arrest.This leads to an increase in patients with hypoxic brain injury (HBI) who are in serious needof rehabilitation. Both hypoxic brain injury and cardiopulmonary arrest can cause many complications.Chronic post-hypoxic myoclonus, also known as Lance and Adams syndrome (LAS), is a rarecondition that occurs after successful cardiopulmonary resuscitation. Here, we present a case withHBI that complicated by spasticity and LAS because of both it is a rare condition and effected to rehabilitationprocess.

Özet

Anafilaksi akut, yaşamı tehdit eden bir durumdur ve sıklıkla ölümle sonuçlanır. Tıbbi gelişmeler ile günümüzde anafilaksiye bağlı kardiorespiratuar arrest sonrasında hastalar daha fazla yaşatılabilmektedir. Bu durum beraberinde ciddi rehabilitasyon ihtiyacı olan hipoksik beyin hasarlı (HBH) hastaların artışını getirmektedir. Hipoksik beyin hasarının kendisi ve oluşan kardiyopulmoner arreste bağlı pek çok komplikasyon gelişebilmektedir. Lance ve Adams Sendromu (LAS) olarak da bilinen kronik post-hipoksik myoklonus, başarılı bir kardiyopulmoner resustasyondan sonra oluşan nadir bir durumdur. Burada spastisite ve LAS sendromu ile komplike olan HBH'lı hasta, nadir bir durum olması ve rehabilitasyonu etkilemesi nedeni ile sunulmuştur.

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Cite This Article

UMAY, E., DELİBAŞ, O., ÇAKCI, A., GÜNDOĞDU, İ. (2022). Treatment Resistance in Hypoxic Brain Injury Caused by Cardiopulmonary Arrest Due to an Allergic Anaphylaxis: Spasticity, Myoclonus or Both?. Fiziksel Tıp ve Rehabilitasyon Bilimleri Dergisi, Cilt: 22 - Sayı: 1, 29–32. https://ajindex.com/articles/516794

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References (2)

  1. Javadi M, Gholami K, Torkamandi H, Hayatshahi A. Penicillin allergy and cross-reactivity with other beta-lactams. J Pharm Care. 2013;1:29-32.
  2. Gruchalla RS, Pirmohamed M. Clinical practice. Antibiotic allergy. N Engl J Med. 2006;354:601-9. [Crossref] [PubMed] Shrestha D, Dhakal AK, Shakya H, et al. A report of near fatal ceftriaxone induced anaphylaxis in a child with review of literature. Nepal Med Coll J. 2013;15:84-6. [PubMed] Wolstenholme N, Moore B. The clinical manifestations of anoxic brain injury. Progress in Neurology and Psychiatry. 2010;14:8-13. [Crossref] Božic K, Gebauer-Bukurov K, Sakalaš L, et al. Improvement of post-hypoxic action myoclonus with levetiracetam add-on therapy: a case report. Vojnosanit Pregl. 2014;71:515-9. [Crossref] [PubMed] Peskine A, Picq C, Pradat-Diehl P. Cerebral anoxia and disability. Brain Inj. 2004;18:1243- 54. [Crossref] [PubMed] CullenNK,WeiszK.Cognitive correlateswith functional outcomes after anoxic brain injury: a casecontrolled comparison with traumatic brain injury. Brain Inj. 2011;25:35-43. [Crossref] [PubMed] Smania N, Avesani R, Roncari L, et al. Factors predicting functional and cognitive recovery following severe traumatic, anoxic, and cerebrovascular brain damage. J Head Trauma Rehabil. 2013;28:131-40. [Crossref] [PubMed] Lance JW, Adams RD. The syndrome of intention or action myoclonus as a sequel to hypoxic encephalopathy. Brain. 1963;86:111- 36. [Crossref] Tai KK, Truong DD. Post-hypoxicmyoclonus induces Fos expression in the reticular thalamic nucleus and neurons in the brainstem. Brain Res. 2005;1059:122-8. [Crossref] [PubMed]