Journals / Ege Tıp Dergisi / 2011 / Cilt: 50 - Sayı: 2

The effect of different intensity walking programs on resistin and visfatin levels in pre-menopausal women

Farklı siddetteki yürüyüs programlarının pre-menopozal kadınlarda resistin ve visfatin düzeyleri üzerine etkisi

Pages
87–94
Publication Date
2022-10-17
DOI
—
🏛️Bibliographic metadata and full text access for this article are provided via DergiPark.
DergiPark Record ↗
✨

AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

Results: VO2max improved in both exercise groups (p

📌 3 Key Takeaways

🎯
Objective & Hypothesis:Aim: The aim of the study was to examine the effect of two different intensity (walking speeds) walking programs on resistin and visfatin levels in pre-menopausal women.
📊
Key Findings & Data:Material and Methods: Thirty-seven pre-menopausal women (30-49 years) were divided into a high-intensity walking group (HIWG; n=12), a moderate-intensity walking group (MIWG; n=14), and a control group (CG; n=11).
💡
Conclusion & Impact:Results: VO2max improved in both exercise groups (p
🔬Design:Modeling & Simulation
📍Scope:Comprehensive Sample
⚙️Variables:Domain Parameters

Abstract

Aim: The aim of the study was to examine the effect of two different intensity (walking speeds) walking programs on resistin and visfatin levels in pre-menopausal women. Material and Methods: Thirty-seven pre-menopausal women (30-49 years) were divided into a high-intensity walking group (HIWG; n=12), a moderate-intensity walking group (MIWG; n=14), and a control group (CG; n=11). The exercise groups walked for twelve weeks, five days per week from 30-60min/day. The HIWG and MIWG walked at 70-75% and 50-55% maximum heart rate reserve, respectively. Body weight, percent body fat, body mass index (BMI), waist circumference (WC), maximal oxygen consumption (VO2max), glucose, insulin, resistin, and visfatin were measured; the HOMA-IR (homeostasis model assessment of insulin resistance) index was calculated before and after the study. Results: VO2max improved in both exercise groups (p

Özet

Amaç: Çalısmanın amacı on iki haftalık, iki farklı siddette (yürüme hızında) yapılan yürüyüs programlarının resistin ve visfatin düzeyleri üzerine etkisini incelemekti. Yöntem ve Gereç: Otuz yedi pre-menopozal kadın (30-49 yas) hızlı tempo yürüyüs grubu (HTYG; n= 12), orta tempo yürüyüs grubu (OTYG; n=14) ve kontrol grubu (KG; n= 11) olarak ayrıldı. Egzersiz grupları 12 hafta, haftada bes gün, günde 30-60 dakika yürüdüler. HTYG ve OTYG maksimum kalp atım sayısı yedeğinin sırasıyla %70-75 ve %50- 55’inde yürüdüler. Antrenman periyodundan önce ve sonra vücut ağırlığı (VA), vücut yağ oranı (VYO), beden kitle indeksi (BKĐ), bel çevresi (BÇ), maksimal oksijen tüketimi (VO2max), glikoz, insülin, resistin ve visfatin düzeyleri ölçüldü; HOMA-IR (insülin direncinin homostatik model değerlendirilmesi) hesaplandı. Bulgular: VO2max her iki egzersiz grubunda (p

🔖

Cite This Article

BÜYÜKYAZI, G., ESEN, H., GÖZLÜKAYA, F., ÖZCAN, İ., TIKIZ, H., ULMAN, C., TANELİ, F. (2022). The effect of different intensity walking programs on resistin and visfatin levels in pre-menopausal women. Ege Tıp Dergisi, Cilt: 50 - Sayı: 2, 87–94. https://ajindex.com/articles/496185

📄 Full Text PDF Viewer

📄

PDF görüntüleyici hazırlanıyor...

References (15)

  1. World Health Organization. Physical status: The use and interpretation of anthropometry. World Health Organ Tech Rep Ser 1995; 854:1-452
  2. Pi-Sunyer FX. Medical hazards of obesity. Ann Intern Med 1993; 119:655-660
  3. Steppan, CM, Bailey ST, Bhat S, et al. The hormone resistin links obesity to diabetes. Nature 2001; 409:307-312
  4. Kawanami D, Maemura K, Takeda N, et al. Direct reciprocal effects of resistin and adiponectin on vascular endothelial cells: A new insight into adipocytokine-endothelial cell interactions. Biochem Biophys Res Commun 2004; 314:415-419
  5. Verma S, Li SH, Wang CH, et al. Resistin promotes endothelial cell activation: Further evidence of adipokine-endothelial interaction. Circulation 2003; 108:736-740
  6. Yang RZ, Huang Q, Xu A, et al. Comparative studies of resistin expression and phylogenomics in human and mouse. Biochem Biophys Res Commun 2003; 310:927-935
  7. Frankel DS, Vasan RS, D’Agostino RB, et al. Resistin, adiponectin, and risk of heart failure: The Framingham Offspring Study. J Am Coll Cardiol 2009; 53(9):754-762
  8. Pischon T, Bamberger CM, Kratzsch J, et al. Association of plasma resistin levels with coronary heart disease in women. Obes Res 2005; 13:1764-1771
  9. Fukuhara A, Matsuda M, Nishizawa M, et al. Visfatin: A protein secreted by visceral fat that mimics the effects of insulin. Science 2005; 307:426-430
  10. Berndt J, Klöting N, Kralish S, et al. Plasma visfatin concentrations and fat depot-specific mRNA expression in humans. Diabetes 2005; 54:2911-2916
  11. Arner P. Visfatin–a true or false trail to type 2 diabetes mellitus. J Clin Endocrinol Metab 2006; 91:28-30
  12. Klöting N, Klöting I. Visfatin: Gene expression in isolated adipocytes and sequence analysis in obese WOKW rats compared with lean control rats. Biochem Biophys Res Commun 2005; 332(4):1070-1072
  13. Pagano C, Pilon C, Oliveri M, et al. Reduced plasma visfatin/pre-B cell colony-enhancing factor in obesity is not related to insulin resistance in humans. J Clin Endocrinol Metab 2006; 91:3165-3170
  14. Knowler WC, Barrett-Connor E, Fowler SE, et al. Diabetes Prevention Program Research Group. Reduction in the incidence of type 2 diabetes with lifestyle intervention or metformin. N Engl J Med 2002; 346:393-403
  15. Tuomilehto J, Lindstrom J, Eriksson JG, et al. Finnish Diabetes Prevention Study Group: prevention of type 2 diabetes mellitus by changes in lifestyle among subjects with impaired glucose tolerance. N Engl J Med 2001; 344:1343-1350. 1