Journals / Türk Göğüs Kalp Damar Cerrahisi Dergisi / 2010 / Cilt: 18 - Sayı: 4

Noninvasive mechanical ventilation in the treatment of acute cardiogenic pulmonary edema

Akut kardiyojenik pulmoner ödem tedavisinde noninvazif mekanik ventilasyon

Pages
341–347
Publication Date
2022-09-02
DOI
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AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

Both CPAP and BIPAP have been demonstrated to be well tolerated and are not associated with any serious events.

📌 3 Key Takeaways

🎯
Objective & Hypothesis:Acute cardiogenic pulmonary edema (ACPE) is a common cause of respiratory failure that necessitates endotracheal intubation and invasive mechanical ventilation.
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Key Findings & Data:In patients with ACPE noninvasive mechanical ventilation (NIMV) may reduce intubation rate and hence, the complications related with endotracheal intubation.
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Conclusion & Impact:Both CPAP and BIPAP have been demonstrated to be well tolerated and are not associated with any serious events.
🔬Design:Research Article
📍Scope:Comprehensive Sample
⚙️Variables:Domain Parameters

Abstract

Acute cardiogenic pulmonary edema (ACPE) is a common cause of respiratory failure that necessitates endotracheal intubation and invasive mechanical ventilation. In patients with ACPE noninvasive mechanical ventilation (NIMV) may reduce intubation rate and hence, the complications related with endotracheal intubation. There has been a steady stream of trials investigating the effectiveness of NIMV in the treatment of ACPE, and NIMV has been shown to improve physiological parameters quickly and decrease endotracheal intubation and mortality. There are essentially two modes of NIMV that are used in the treatment of ACPE: continuous positive airway pressure (CPAP) and noninvasive pressure support ventilation or bilevel positive airway pressure (BIPAP). Both CPAP and BIPAP have been demonstrated to be well tolerated and are not associated with any serious events.

Özet

Akut kardiyojenik pulmoner ödem (AKPÖ), endotrakeal entübasyon ve invazif mekanik ventilasyon gereksinimin gelişebildiği, solunum yetmezliğinin sık bir nedenidir. Noninvazif mekanik ventilasyon (NİMV) AKPÖ'lü hastalarda entübasyon oranlarını ve böylece endotrakeal entübasyon ile ilişkili komplikasyonları azaltabilir. Akut kardiyojenik pulmoner ödem tedavisinde NİMV'nin etkinliğini değerlendiren gittikçe artan sayıda çalışma bulunmaktadır ve NİMV'nin fizyolojik parametreleri hızla düzelttiği, endotrakeal entübasyon ve mortaliteyi azalttığı gösterilmiştir. Noninvazif mekanik ventilasyonun AKPÖ tedavisinde uygulanan başlıca iki modu bulunmaktadır: sürekli pozitif havayolu basıncı (CPAP) ve noninvazif basınç destekli ventilasyon veya bilevel pozitif havayolu basıncı (BİPAP). Hem CPAP hem de BİPAP'ın iyi tolere edildiği ve ciddi yan etkiye neden olmadıkları gösterilmiştir.

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Cite This Article

KAYA, A., ÇİLEDAĞ, A. (2022). Noninvasive mechanical ventilation in the treatment of acute cardiogenic pulmonary edema. Türk Göğüs Kalp Damar Cerrahisi Dergisi, Cilt: 18 - Sayı: 4, 341–347. https://ajindex.com/articles/424960

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References (13)

  1. Gheorghiade M, Zannad F, Sopko G, Klein L, Piña IL, Konstam MA, et al. Acute heart failure syndromes: current state and framework for future research. Circulation 2005; 112:3958-68
  2. Zannad F, Adamopoulos C, Mebazaa A, Gheorghiade M. The challenge of acute decompensated heart failure. Heart Fail Rev 2006;11:135-9
  3. Gray A, Schlosshan D, Elliott MW. NIV for cardiogenic pulmonary oedema. Eur Respir Mon 2008;41:72-93
  4. Nieminen MS, Brutsaert D, Dickstein K, Drexler H, Follath F, Harjola VP, et al. EuroHeart Failure Survey II (EHFS II): a survey on hospitalized acute heart failure patients: description of population. Eur Heart J 2006;27:2725-36
  5. Girou E, Brun-Buisson C, Taillé S, Lemaire F, Brochard L. Secular trends in nosocomial infections and mortality associated with noninvasive ventilation in patients with exacerbation of COPD and pulmonary edema. JAMA 2003;290:2985-91
  6. Gürkan ÖU, Berk Ö, Kaya A, Çelik G, Kumbasar Ö, Acıcan T, et al. Evaluation of a respiratory intermediate care unit in Ankara: Two year analysis. Turkish Respir J 2001;2:20-25
  7. Keenan SP, Sinuff T, Cook DJ, Hill NS. Which patients with acute exacerbation of chronic obstructive pulmonary disease benefit from noninvasive positive-pressure ventilation? A systematic review of the literature. Ann Intern Med 2003;138:861-70
  8. Masip J, Roque M, Sánchez B, Fernández R, Subirana M, Expósito JA. Noninvasive ventilation in acute cardiogenic pulmonary edema: systematic review and meta-analysis. JAMA 2005;294:3124-30
  9. Peter JV, Moran JL, Phillips-Hughes J, Graham P, Bersten AD. Effect of non invasive positive pressure ventilation (NIPPV) on mortality in patients with acute cardiogenic pulmonary oedema: a meta-analysis. Lancet 2006;367:1155-63
  10. Winck JC, Azevedo LF, Costa-Pereira A, Antonelli M, Wyatt JC. Efficacy and safety of non-invasive ventilation in the treatment of acute cardiogenic pulmonary edema-a systematic review and meta-analysis. Crit Care 2006;10:R69
  11. Garpestad E, Brennan J, Hill NS. Noninvasive ventilation for critical care. Chest 2007;132:711-20
  12. Bulkley BH, Hutchins GM, Bailey I, Strauss HW, Pitt B. Thallium 201 imaging and gated cardiac blood pool scans in patients with ischemic and idiopathic congestive cardiomyopathy. A clinical and pathologic study. Circulation 1977;55:753-60
  13. Naughton MT, Rahman MA, Hara K, Floras JS, Bradley TD. Effect of continuous positive airway pressure on intrathoracic and left ventricular transmural pressures in patien