SavedAdd your journal

Journals / Şişli Etfal Hastanesi Tıp Bülteni / 2018 / Cilt: 52 - Sayı: 2

Comparison of Postoperative Smoking Cessation Rates of Patients with Benign or Premalignant Vocal Cord Lesions

Bilge TÜRK
Meltem AKPINAR
Senem Kurt DİZDAR
Kerem Sami KAYA
Alican Çoktur
BERNA USLU COŞKUN
Pages
114–118
DOI
—
🏛️Bibliographic metadata and full text access for this article are provided via DergiPark.
DergiPark Record ↗
✨

AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

Premalignant vocal cord lesions require clinical follow-up and treatment because of the risk and potential for their transformation into in situ or invasive laryngeal carcinomas.

📌 3 Key Takeaways

🎯
Objective & Hypothesis:Objectives: It has been determined that cigarette is a risk factor for squamous cell carcinomas of the oral cavity, esophagus, and larynx.
📊
Key Findings & Data:We aimed to investigate the role of histopathological diagnosis of the lesion in smoker patients with vocal cord lesion on smoking cessation rates and to determine strategies to help them quit smoking.
💡
Conclusion & Impact:Premalignant vocal cord lesions require clinical follow-up and treatment because of the risk and potential for their transformation into in situ or invasive laryngeal carcinomas.
🔬Design:Research Article
📍Scope:2014 Verileri
⚙️Variables:Domain Parameters

Özet

Objectives: It has been determined that cigarette is a risk factor for squamous cell carcinomas of the oral cavity, esophagus, and larynx. We aimed to investigate the role of histopathological diagnosis of the lesion in smoker patients with vocal cord lesion on smoking cessation rates and to determine strategies to help them quit smoking. Methods: In this prospective clinical study, we included 182 (112 male, 70 female) smoker patients who underwent direct laryngoscopy and biopsy due to premalignant (dysplasia) or benign (polyp, leukoplasia, nodule) vocal cord lesions between July 2014 and December 2017 at our clinic. Smoking habits (ex-smoker, current smoker) of all smoker patients were questioned at least 6 months postoperatively, and postoperative smoking cessation rates were compared. Results: When the smoking cessation rates of the patients with benign and premalignant vocal cord lesions were evaluated, the smoking cessation rate of the patients with premalignant vocal cord lesions was 3.45 times higher than that of the patients with benign vocal cord lesions (OR, 3.45; 95% CI, 1.76–6.74) (p<0.001). The postoperative application rate of the patients to smoking cessation outpatient clinics was low (6%). Male patients with premalignant lesions were more likely to quit smoking than female patients (p=0.001). Conclusion: Patients with premalignant vocal cord lesions had higher smoking cessation rates. Premalignant vocal cord lesions require clinical follow-up and treatment because of the risk and potential for their transformation into in situ or invasive laryngeal carcinomas.

🔖

Cite This Article

TÜRK, B., AKPINAR, M., DİZDAR, S. K., KAYA, K. S., Çoktur, A., COŞKUN, B. U. (2018). Comparison of Postoperative Smoking Cessation Rates of Patients with Benign or Premalignant Vocal Cord Lesions. Şişli Etfal Hastanesi Tıp Bülteni, Cilt: 52 - Sayı: 2, 114–118. https://ajindex.com/articles/364020

📄 Full Text PDF Viewer

📄

PDF görüntüleyici hazırlanıyor...

References (9)

  1. Argüder E, Karalezli A, Hezer H, Kılıç H, Er M, Hasanoğlu HC et al. Factors Affecting the Success of Smoking Cessation. Turk Toraks Derg 2013;14:81–7.
  2. West R, Raw M, McNeill A, Stead L, Aveyard P, Bitton J, et al. Healthcare interventions to promote and assist tobacco cessation: a review of efficacy, effectiveness and affordability for use in national guideline development. Addiction 2015;110:1388–403.
  3. Fidler J, Ferguson SG, Brown J, Stapleton J, West R. How does rate of smoking cessation vary by age, gender and social grade? Findings from a population survey in England. Addiction 2013;108:1680–5.
  4. Van Schayck OCP, Williams S, Barchilon V, Baxter N, Jawad M, Katsaounou PA, et al. Treating tobacco dependence: guidance for primary care on life-saving interventions. Position statement of the IPCRG. NPJ Prim Care Respir Med 2017;27:38. Norberg M, Lundqvist G, Nilsson M, Gilljam H, Weinehall L. Changing patterns of tobacco use in a middle-aged population: the role of snus, gender, age, and education. Glob Health Action 2011;4. Farmer MM, Rose DE, Riopelle D, Lanto AB, Yano EM. Gender differences in smoking and smoking cessation treatment: an examination of the organizational features related to care. Womens Health Issues 2011;21:S182–9. Sadri M, McMahon J, Parker A. Laryngeal dysplasia: aetiology and molecular biology. J Laryngol Otol 2006;120:170–7. Thibeault SL. Advances in our understanding of the Reinke space. Curr Opin Otolaryngol Head Neck Surg 2005;13:148–51.
  5. Wu CC, Chen MC, Huang YK, Huang CY, Lai LA, Chung CJ, et al. Environmental tobacco smoke and arsenic methylation capacity are associated with urothelial carcinoma. J Formos Med Assoc 2013;112:554–60.
  6. De Flora S, Izzotti A, D'Agostini F, La Maestra S, Micale RT, Ceccaroli C, et al. Rationale and approaches to the prevention of smoking-related diseases: overview of recent studies on chemoprevention of smoking-induced tumors in rodent models. J Environ Sci Health C Environ Carcinog Ecotoxicol Rev 2014;32:105–20.
  7. Blot WJ, McLaughlin JK, Winn DM, Austin DF, Greenberg RS, Preston-Martin S, et al. Smoking and drinking in relation to oral and pharyngeal cancer. Cancer Res. 1988 Jun 1;48(11):3282-7.
  8. Franceschi S, Talamini R, Barra S, Barón AE, Negri E, Bidoli E, et al. Smoking and drinking in relation to cancers of the oral cavity, pharynx, larynx, and esophagus in northern Italy. Cancer Res 1990;50:6502–7.
  9. Demir M, Karadeniz G, Demir F, Karadeniz C, Kaya H, Yenibertiz D, et al. The impact of anti-smoking law