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Journals / Van Tıp Dergisi / 2006 / Cilt: 13 - Sayı: 1

Cor pulmonale due to adenotonsillar hypertrophy in a child

Çocukta adenotonsiller hipertrofiye bağlı kor pulmonale

A. Faruk KIROĞLU
Hakan ÇANKAYA
Abdurrahman ÜNER
Köksal YUCA
Rezan OKYAY
Pages
19–21
DOI
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AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

In this case report we want to present a child with cor pulmonale secondary to adenotonsiliar hypertrophy which was successfully treated with surgial removal.

📌 3 Key Takeaways

🎯
Objective & Hypothesis:The most common cause of pediatric airway obstruction is the hypertrophy of tonsils and adenoids.
📊
Key Findings & Data:If the obstruction to upper airways is not relieved, the child can develop obstructive sleep apnea (OSA) and its complications.
💡
Conclusion & Impact:In this case report we want to present a child with cor pulmonale secondary to adenotonsiliar hypertrophy which was successfully treated with surgial removal.
🔬Design:Research Article
📍Scope:Comprehensive Sample
⚙️Variables:Hipertrofi · Çocuk · Uyku apne

Abstract

The most common cause of pediatric airway obstruction is the hypertrophy of tonsils and adenoids. If the obstruction to upper airways is not relieved, the child can develop obstructive sleep apnea (OSA) and its complications. Cardiovascular impairment including cor pulmonale and right heart failure are the possible Ufe-threating complications in OSA. In this case report we want to present a child with cor pulmonale secondary to adenotonsiliar hypertrophy which was successfully treated with surgial removal.

Özet

Pediatrik havayolu tıkanıklıklarının en sık sebebi tonsillerin ve adenoidin büyümüş olmasıdır. Üst solunum yollarındaki tıkanıklık giderilmez ise çocukta obstriiktif uyku apnesî (OUA) ve buna bağlı komplikasyonlar gelişebilir. Kor pulmonale ve sağ kalp yetmezliği OUA'nın yaşamı tehdit edebilecek kardiyovasküler komplikasyonlardır. Bu olgu sunumunda adenotonsiller hipertrofiye bağlı olarak gelişen ve cerrahi eksizyon ile tedavi edilen bir pediyatrik kor pulmonale vakasını sunmak istiyoruz.

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Cite This Article

KIROĞLU, A. F., ÇANKAYA, H., ÜNER, A., YUCA, K., OKYAY, R. (2006). Cor pulmonale due to adenotonsillar hypertrophy in a child. Van Tıp Dergisi, Cilt: 13 - Sayı: 1, 19–21. https://ajindex.com/articles/334737

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References (14)

  1. Guilleminault C, Pelayo R, Leger D, Clerk A, Bocian RC. Recognition of sleep-disordered breathing in children. Pediatrics. 1996;98(5):871-82
  2. Blum RH, McGowan FX Jr. Chronic upper airway obstruction and cardiac dysfunction: anatomy, pathophysiology and anesthetic implications. Paediatr Anaesth. 2004 Jan; 14(1):75-83
  3. Wilkinson AR, McCormick MS, Freeland AP,Pickering D. Electrocardiographic signs of pulmonary hypertension in children who snore. Br Med J (Clin Res Ed). 1981; 282:1579-81
  4. Gorur K, Doven O, Unal M, Akkus N, Ozcan C.Preoperative and postoperative cardiac and clinical findings of patients with adenotonsiliar hypertrophy. Int J Pediatr Otorhinolaryngol. 2001 May 31;59:41-6
  5. Shintani T, Asakura K, Kataura A. The effect of adenotonsillectomy in children with OSA.Int J Pediatr Otorhinolaryngol. 1998;44:51-8
  6. Aji DY, Sarioglu A, Sever L, Arisoy N. Pulmonary hypertension due to chronic upper airway obstruction: a clinical review and report of four cases. Turk J Pediatr. 1991;33(1):35-41
  7. Li AM, Hui S, Wong E, Cheung A, Fok TF.Obstructive sleep apnoea in children with adenotonsiliar hypertrophy: prospective study. Hong Kong Med J.;7(3):236-40
  8. Miman MC, Kirazli T, Ozyurek R. Doppler echocardiography in adenotonsiliar hypertrophy. Int J Pediatr Otorhinolaryngol. 2000;54(1):21-6
  9. Sie KC, Perkins JA, Clarke WR. Acute right hearc failure due to adenotonsiliar hypertrophy. Int J Pediatr Otorhinolaryngol. 1997;41(1):53-8
  10. Ramakrishna S, Ingle VS, Patel S, Bhat P, Dada JE,Shah FA, Murty PS. Reversible cardio-pulmonary changes due to adeno4onsilar hypertrophy. Int J Pediatr Otorhinolaryngol. 2000;55(3):203-6
  11. Barst RJ, Rubin LJ, Long WA, McGoon MD, Rich S,Badesch DB, Groves BM, Tapson VF, Bourge RC? Brundage BH, et al. A comparison of continuous intravenous eppprostenol (prostacyclin) with conventional therapy for primary pulmonary hypertension. The Primary Pulmonary Hypertension Study Group. N Engl J Med. 1996;334(5):296-302
  12. Clarke WR, Uezono S, Chambers A, Doepfner P. The type III phosphodiesterase inhibitor milrinone and type V PDE inhibitor dipyridamole individually and synergistically reduce elevated pulmonary vascular resistance. Pulm Pharmacol. 1994;7(2):81-9
  13. Rich S, Kaufmann E, Levy PS. The effect of high doses of calcium-channel blockers on survival in primary pulmonary hypertension. N Engl J Med. 1992;327(2):76-81
  14. Rosen GM, Muckle RP, Mahowald MW, Goding GS,Ullevig C. Postoperative respiratory comprom