SavedAdd your journal

Journals / Medical Network Klinik Bilimler ve Doktor / 2001 / Cilt: 7 - Sayı: 2

Acute peripheral facial palsy secondary to rubella infection: A case report

Kızamıkçığa bağlı akut periferik fasiyal paralizi: Bir olgu sunumu

Tamer KUYUCU
Sefer KUMANDAŞ
Mustafa AKÇAKUŞ
Hakan GÜMÜŞ
Pages
213–215
DOI
—
🏛️Bibliographic metadata and full text access for this article are provided via DergiPark.
DergiPark Record ↗
✨

AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

We emphasize that acute peripheral facial palsy secondary to rubella infection is very rare and rubella should be considered as an etiologic agent of acute peripheral facial palsy.

📌 3 Key Takeaways

🎯
Objective & Hypothesis:The causes of acute peripheral facial palsy (APFP) remain obscure in many patients.
📊
Key Findings & Data:Many viral infections such as herpes virus, Epstein-Barr virus(EBV), mumps virus, parvovirus B19, coxsackie B4 virus, Borrelia burgdorferi, human immunodeficiency virus (HIV) and very rarely rubella virus may play a role in etiology.
💡
Conclusion & Impact:We emphasize that acute peripheral facial palsy secondary to rubella infection is very rare and rubella should be considered as an etiologic agent of acute peripheral facial palsy.
🔬Design:Research Article
📍Scope:Comprehensive Sample
⚙️Variables:Kızamıkçık · Yüz felci · Çocuk

Abstract

The causes of acute peripheral facial palsy (APFP) remain obscure in many patients. Many viral infections such as herpes virus, Epstein-Barr virus(EBV), mumps virus, parvovirus B19, coxsackie B4 virus, Borrelia burgdorferi, human immunodeficiency virus (HIV) and very rarely rubella virus may play a role in etiology. A 15-month-old boy with the complaints of withdrawal of the mouth to the right while crying, unable to close his left eye, and swelling of the neck was seen in our outpatient clinic. On his examination left acute peripheral facial palsy, retroauricular, posterior cervical and suboccipital lymph nodes were detected. Serologic evaluation confirmed the diagnosis of rubella. Serological and laboratory tests were negative for other etiologic agents. In follow up the patient, lymph nodes subsided within two weeks and facial paralysis recovered within eight weeks without any treatment. We emphasize that acute peripheral facial palsy secondary to rubella infection is very rare and rubella should be considered as an etiologic agent of acute peripheral facial palsy.

Özet

Akut periferik fasiyal paralizinin (APFP) etyolojisi tam aydınlatılamamış olup, etiyolojide sorumlu tutulan enfeksiyöz ajanlar arasında herpes zoster virus, Epstein Barr virus, herpes simplex virus , kabakulak virusu, coxsackie B4, Borrelia burgdorferi, human immune deficiency virus , parvovirus B19 ve nadirende kızamıkçık virusu sayılmaktadır. Ağlarken ağız kenarında sağa çekilme, sol gözünü kapatamama, her iki kulak arkasında ve boyunda şişlik şikayeti ile polikliniğimize getirilip, fizik muayenesinde sol akut periferik fasiyal paralizisi, bilateral servikal ve submandibuler lenfadenopatisi tesbit edilen onbeş aylık erkek hastanın laboratuar tetkiklerinde rubella serolojisi pozitifliği dışında etiyolojiyi açıklayabilecek başka neden bulunamadı. APFP'sinin kızamıkçığa sekonder geliştiği düşünüldü. Hiçbir tedavi verilmeyen hastanın ikinci haftanın sonunda lenfadenopatilerinin küçüldüğü, sekizinci haftada paralizinin tam olarak düzeldiği ve lenfadenopatilerinin kaybolduğu gözlendi. Kızamıkçığa bağlı APFP'nin nadir olması ve APFP'nin etyolojisi araştırılırken kızamıkçığın da düşünülmesi gerektiğinin vurgulanması amacıyla olgunun sunulmasına karar verildi.

🔖

Cite This Article

KUYUCU, T., KUMANDAŞ, S., AKÇAKUŞ, M., GÜMÜŞ, H. (2001). Acute peripheral facial palsy secondary to rubella infection: A case report. Medical Network Klinik Bilimler ve Doktor, Cilt: 7 - Sayı: 2, 213–215. https://ajindex.com/articles/278686

References (11)

  1. Yanagihara N. Incidence of bell's palsy. Ann Otol Rhinol Laryngol 1988; 97:3-4.
  2. Morgan M, Nathwani D. Facial palsy and infection: the unfolding story.Clin Infect Dis 1992; 14:263-71.
  3. Shaw FE, Graham DJ, Guess HA, et al. Postmarketing surveillance for neurologic adverse events reported after hepatitis B vaccination. Experience of the first three years. Am J Epidemiol. 1988; 127:337-52. Kohler A. Isolated peripheral facial paralysis and HIV infection: 7 cases. Rev Neurol (Paris). 1995; 151:332-7.
  4. Jamal GA, AL - Husami A. Bell's palsy and infection with rubella virus. J Neurol Neurosurg Psychiatry. 1983; 46:678-80.
  5. Martinon F- Torres F, Seara M J F, Pastoriza I D R. Parvovirus B19 infection complicated by peripheral facial palsy and parotitis with intraparotid lymphadenitis. Pediatr Infect Dis J. 1999; 18:307-8. Matthews W B. The treatment of Bell's palsy. Recent Advances in Clinical Neurology 1982; 3:239-48. Thomas PK. Bell's palsy In: Weatherall DJ, Ledingham JGG, Warrel DA,eds. Oxford textbook of medicine. 2nd ed. Vol. 2. oxford:oxford University Press. 1988; 94-5.
  6. Ghonim MR, Gavilan C, Sarri M J.Bilateral similtaneous Bell's palsy. Two cases following herpes simplex gingivostomatitis.ORL.J Otorhinolaryngol relat Spec 1988; 50:269-72.
  7. Hadar T, Tovi F, Sidi J, et al. Specific IgG and IgA antibodies to herpes simplex virus and varicella zoster virus in acute peripheral facial palsy patients. J Med Virol. 1983; 12:237-45.
  8. Clark JA, Carlson RD, Sasaki CT, et al. Facial paralysis in Lyme Disease. Laryngoscope 1985; 95:1341-5. Fowler E P. The pathologic findings in a case of facial paralysis. Trans Am Acad Ophthalmol Otolaryngol 1963; 67:187-97.
  9. Nieuwmeyer PA, Visser SL, Feenstra L. Bell's palsy:a polyneuropathy. Am J Otolaryngol 1985; 6:250-2.
  10. Djupesland G, Berdal P, Johannessen TA, et al. Acute peripheral facial palsy. Part of a cranial polyneuropathy. Arch Otolaryngol 1977; 103:641-4.
  11. Hyden D, Roberg M, Forsberg P, et al. Acute "idiopathic" peripheral facial palsy: clinical, serological, and cerebrospinal fluid findings and effects of corticosteroids. Am J Otolaryngol. 1993; 14:179-86.