Journals / Fırat Tıp Dergisi / 2018 / Cilt: 23 - Sayı: 0

Obesity and Endocrine Syst

Obezite ve Endokrin Sistem

Pages
14–17
DOI
—
🏛️Bibliographic metadata and full text access for this article are provided via DergiPark.
DergiPark Record ↗
✨

AI Summary & Key Takeaways

AI Insight
⚡ TL;DR(Too Long; Didn't Read)

In conclusion, obesity, a major problem with multifactorial etiology has become an epidemy worldwi-de.

📌 3 Key Takeaways

🎯
Objective & Hypothesis:Being associated with serious complications, obesity deteriorates the quality of life and causes increase in rates of early death.
📊
Key Findings & Data:Insulin resistance, metabolic syndrome, prediabe-tic state, type 2 diabetes mellitus, dyslipidemia, hypertension, policystic ovary syndrome and problems regarding fertility are possible results of obesity associated with the endocrine system.
💡
Conclusion & Impact:In conclusion, obesity, a major problem with multifactorial etiology has become an epidemy worldwi-de.
🔬Design:Research Article
📍Scope:Comprehensive Sample
⚙️Variables:Domain Parameters

Abstract

Being associated with serious complications, obesity deteriorates the quality of life and causes increase in rates of early death. Since it leads to syste-mic problems, particularly metabolic abnormalities, obesity is one of the major public health issues. Insulin resistance, metabolic syndrome, prediabe-tic state, type 2 diabetes mellitus, dyslipidemia, hypertension, policystic ovary syndrome and problems regarding fertility are possible results of obesity associated with the endocrine system. In conclusion, obesity, a major problem with multifactorial etiology has become an epidemy worldwi-de. In order to prevent obesity, governments need to set policies. The collaboration of food industry and media is considered to be very important in preventing this major health problem.

Özet

Obezite ciddi metabolik komplikasyonlara neden olurken, hayat kalitesini bozmakta ve erken ölüm artışlarına yol açmaktadır. Başta kardiyometabolik hastalıklar olmak üzere diğer sistemik anormalliklerin ortaya çıkışını kolaylaştıran önemli bir halk sağlığı problemidir. Endokrin sistemi ilgilendiren başlıca problemler, insülin direnci, metabolik sendrom, prediyabet, tip 2 diabetes mellitus, dislipidemi, hipertansiyon, polikistik over sendromu ve fertilite sorunlarıdır. Sonuç olarak; etyolojisinde multifaktöryel nedenler bulunan obezite dünyada epidemi haline gelmiştir. Obezitenin önlenmesi ulusal politikalar sayesinde yasal düzenlenmeler yapılarak ayrıca medya, gıda ve sanayi işbirliği yardımıyla gerçekleşeceği düşünülmektedir.

🔖

Cite This Article

UĞUR, K. (2018). Obesity and Endocrine Syst. Fırat Tıp Dergisi, Cilt: 23 - Sayı: 0, 14–17. https://ajindex.com/articles/164912

📄 Full Text PDF Viewer

📄

PDF görüntüleyici hazırlanıyor...

References (11)

  1. Adams KF, Schatzkin A, Harris TB, et al. Overweight, obesity, and mortality in a large prospective cohort of persons 50 to 71 years old. N Engl J Med 2006; 355: 763-78.
  2. Lemieux I, Pascot A, Couillard C, et al. Hypert-riglyceridemic waist: A marker of the atheroge-nic metabolic triad (hyperinsulinemia; hypera-polipoprotein B; small, dense LDL) in men? Circulation 2000; 102: 179-84.
  3. Ruderman N, Chisholm D, Pi-Sunyer X, et al. The metabolically obese, normal-weight indivi-dual revisited. Diabetes 1998; 47: 699-713.
  4. Meigs JB, D’Agostino RB, Wilson WF, et al. Risk variable clustering in the insülin resistance syndrome. Diabetes 1997; 46: 1594-1600.
  5. Janssen I, Katzmarzyk PT, Ross R. Waist cir-cumference and not body mass index explains obesity-related health risk. Am J Clin Nutr 2004; 79: 379. Krssak M, Falk Petersen K, Dresner A, et al. Intramyocellular lipid concentrations are corre-lated with insulin sensitivity in humans: a 1H NMR spectroscopy study. Diabetologia 1999; 42: 113-6.
  6. Perry RJ, Samuel VT, Petersen KF, et al. The role of hepatic lipids in hepatic insulin resistan-ce and type 2 diabetes. Nature 2014; 510: 84-91.
  7. Ibrahim M, Tuomilehto J, Aschner P, et al. Global status of diabetes prevention and pros-pects for action: A consensus statement Diabe-tes Metab Res Rev. 2018 May 14:e3021. doi: 10.1002/dmrr.3021. [Epub ahead of print]. Lam DW, LeRoith D. The worldwide diabetes epidemic. Curr Opin Endocrinol Diabetes Obes 2012; 19: 93-6.
  8. Faerch K, Borch-Johnsen K, Holst JJ, et al. Pathophysiology and aetiology of impaired fas-ting glycaemia and impaired glucose tolerance: does it matter for prevention and treatment of type 2 diabetes? Diabetologia 2009; 52: 1714-23.
  9. Collins R, Reith C, Emberson J et al. Interpreta-tion of the evidence for the effi cacy and safety of statin therapy. Lancet 2016; 388: 2532–61. Ligthart S, van Herpt TT, Leening MJ, et al. Lifetime risk of developing impaired glucose metabolism and eventual progression from pre-diabetes. Lancet Diabetes Endocrin 2016; 4: 44-51.
  10. Propst M, Colvin C, Griffin RL, et al. Diabetes and prediabetes are significantly higher in mor-bidly obese children compared with obese child-ren. Endocr Pract 2015; 21: 1046-53.
  11. Boulton AJ, Armstrong DG, Albert SF, et al. Comprehensive foot examination and risk as-sessment: a report of the task force of the foot care interest group of the American Diabetes Association, with endorsement by the American Association of Clinical Endocrinologists. Diabe-tes Care 2008; 31: 1